Results 161 to 170 of about 2,298 (198)
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The Natural Progression of Synkinesis
Plastic & Reconstructive SurgeryBackground: The severity of postparalysis facial synkinesis typically worsens after onset, but its progression over time is not well-characterized. This study examines the time course of synkinesis progression. Methods: Patients with a
Benjamin, Rail +6 more
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Lagophthalmos in Acquired Oculomotor Synkinesis
American Journal of Ophthalmology, 1984Two consecutive patients who exhibited classic signs of unilateral acquired oculomotor synkinesis demonstrated an inability to appose eyelid margins on the affected side, when attempting gentle closure of the eyes. In one patient, this occurred in conjunction with a unilateral upper eyelid crease on the affected side, and in the other patient it was ...
A B, Safran, A, Roth, J, Berney
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Patterns of III nerve synkinesis
Strabismus, 1998PURPOSE Various patterns of synkinesis have been observed following both congenital and acquired III nerve palsies. Aberrant regeneration is the most widely accepted mechanism which explains this synkinesis. Two models of aberrant regeneration have been proposed, namely central collateral axon sprouting, and misdirection of regenerating neurones at the
, Shuttleworth +3 more
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Acquired abducens-trigeminal synkinesis
Neurology, 1998Acquired synkinesis after trauma affecting the oculomotor nerve was first described by Gowers in 1879.1 Several nerve combinations have been described, including congenital trigemino-oculomotor and trigemino-abducens synkinesis.2-4 We describe a patient with an acquired synkinesis of painful involuntary jaw closure triggered by voluntary leftward gaze.
L M, Shulman, B V, Gallo, W J, Weiner
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Patterns of Facial Nerve Synkinesis
The Laryngoscope, 1996AbstractFacial nerve synkinesis is an extremely distressing sequela of facial paralysis, and its prevention and treatment are less than adequate. Improved therapeutic interventions await better understanding of its pathogenesis.In this study, 11 patients with synkinesis and 11 normal control subjects were quantitatively assessed using a new computer ...
C J, Moran, J G, Neely
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Predictors of Facial Synkinesis Severity
Plastic & Reconstructive SurgeryBackground: There is a lack of studies exploring factors that influence synkinesis severity. This study aimed to identify factors that may contribute to variations in severity of facial synkinesis. Methods: The records of patients with a diagnosis of facial synkinesis presenting ...
Benjamin, Rail +6 more
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Congenital Trochlear–Oculomotor Synkinesis
Journal of Neuro-Ophthalmology, 2014Synkinesis of the extraocular muscles forms a subset of congenital ocular motility abnormalities termed congenital cranial dysinnervation disorders. Synkinesis most frequently involves the abducens or oculomotor nerves and rarely the trochlear nerve. Only 3 such patients have been described in the literature.
Lulu L C D, Bursztyn, Inas, Makar
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Bilateral facial synkinesis in leprosy
BMJ Case Reports, 2012Leprosy is an important cause of cranial nerve palsy in endemic areas where it may be seen in upto 17.6% patients. The authors herein describe a rare case of bilaterally symmetrical facial synkinesis with video documentation and modified blink reflex.
Hardeep Singh, Malhotra +6 more
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A Rare Phenomenon: Oculonasal Synkinesis
Aesthetic Plastic Surgery, 2014Oculonasal synkinesis is the simultaneous contraction of the orbicularis oculi and the compressor narium minor muscles. The etiology of this phenomenon is still unclear; congenital and traumatic reasons are considered to be responsible. Here we report a case of oculonasal synkinesis.
N Sinem, Ciloglu +2 more
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Ugeskrift for Læger
Synkinesis is a sequela of moderate to severe facial palsy, caused by aberrant nerve regeneration. It leads to involuntary co-movements, impaired facial expression, and thus reduced quality of life. Treatment includes facial therapy, EMG biofeedback, botulinum toxin (BTX-A), and, in selected cases, surgery.
Cecilie, Jansen +2 more
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Synkinesis is a sequela of moderate to severe facial palsy, caused by aberrant nerve regeneration. It leads to involuntary co-movements, impaired facial expression, and thus reduced quality of life. Treatment includes facial therapy, EMG biofeedback, botulinum toxin (BTX-A), and, in selected cases, surgery.
Cecilie, Jansen +2 more
openaire +2 more sources