Results 311 to 320 of about 3,434,192 (362)

Systemic Inflammation

Encyclopedia of Molecular Pharmacology, 2021
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Inflammation in the nervous system

Current Opinion in Neurobiology, 1995
Earlier studies on inflammation in the CNS have largely focused on conditions with an immune component. Recent evidence has emerged, however, that the innate, acute inflammatory response in the CNS parenchyma is quite unlike that in other tissues. The meninges and ventricular compartments show more typical responses, as does the parenchyma of the brain
V H, Perry, M D, Bell, H C, Brown, M K, Matyszak   +3 more
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PTSD, Immune System, and Inflammation

2023
Posttraumatic stress disorder (PTSD) is a severe trauma and stress-related disorder associated with different somatic comorbidities, especially cardiovascular and metabolic disorders, and with chronic low-grade inflammation. Altered balance of the hypothalamic-pituitary-adrenal (HPA) axis, cytokines and chemokines, C-reactive protein, oxidative stress ...
Nela, Pivac, Barbara, Vuic, Marina, Sagud, Gordana, Nedic Erjavec, Matea, Nikolac Perkovic, Marcela, Konjevod, Lucija, Tudor, Dubravka, Svob Strac, Suzana, Uzun, Oliver, Kozumplik, Sandra, Uzun, Ninoslav, Mimica   +11 more
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Systemic inflammation in the elderly

Best Practice & Research Clinical Anaesthesiology, 2011
Inflammation is an adaptive response to surgery. When the pro-inflammatory responses are unregulated and become over reactive, systemic inflammatory response syndrome may occur. Postoperative systemic inflammation is more common than is generally acknowledged and is observed in about 10-15% of elderly patients undergoing major surgery.
Emily G Y, Koo, Linda M L, Lai, Gordon Y S, Choi, Matthew T V, Chan   +3 more
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The systemic response to inflammation

Biochemical Pharmacology, 1968
Abstract Various acute phase reactants (APR) have been studied in rats with different types of inflammation. These APR (serum albumin, serum globulins, plasma fibrinogens, serum alpha-2-glycoprotein [GP], total serum glycoproteins, serum mucoproteins) and neutrophils and lymphocytes reflect the onset, intensity, and duration of many types of ...
E M, Glenn, B J, Bowman, T C, Koslowske
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The Complement System and Inflammation

1979
The complement system was discovered toward the end of the nineteenth century as a heat-labile principle in fresh nonimmune serum which was essential for the bactericidal activity of serum from immunized guinea pigs. It has subsequently been established as a very important factor of constitutive immunity. The complement system functions as an auxiliary
J O, Minta, H Z, Movat
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Systemic Inflammation Response Index and Systemic Immune-inflammation Index for Predicting the Prognosis of Patients with Aneurysmal Subarachnoid Hemorrhage.

Journal of Stroke & Cerebrovascular Diseases, 2021
OBJECTIVES Inflammatory response plays a pivotal role in the progress of aneurysmal subarachnoid hemorrhage (aSAH). As novel inflammatory markers, systemic inflammation response index (SIRI) and systemic immune-inflammation (SII) index could reflect ...
Seonyong Yun, H. Yi, D. H. Lee, J. Sung
semanticscholar   +1 more source

Apelin/APJ system in inflammation

International Immunopharmacology, 2022
Apelin, an endogenous ligand for the G protein-coupled receptor (APJ), is widely distributed within the central nervous system and diverse organs in human and animals. Recent studies indicate that the apelin/APJ system plays an important role in physiological and pathophysiological situations.
Xu Wang, Lu Zhang, Peisen Li, Yafei Zheng, Yanjie Yang, Shaoping Ji   +5 more
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Systems Biology and Inflammation

2010
Inflammation is a complex, multiscale biological response to threats - both internal and external - to the body, which is also required for proper healing of injured tissue. In turn, damaged or dysfunctional tissue stimulates further inflammation.
Yoram, Vodovotz, Gary, An
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Central nervous system inflammation

European Journal of Anaesthesiology, 2008
Activation of inflammation is the hallmark of pathological processes that follow acute injury. This process is mediated by inflammatory cytokines and adhesion molecules that reside on the surface of endothelium, leucocyte and inflammatory cells. Attenuation of the adhesion cascade has been the subject of several basic science and clinical trials in the
S G, Soriano, S, Piva
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