Results 311 to 320 of about 107,776 (362)
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2012
In the last two decades our understanding of MTLE and its pathophysiology has grown remarkably. Perhaps the most important recognition is that it is not a single entity with a uniform pathology. Rather, it is associated with significant variations in pathology that, in turn, are likely associated with different causes, functional anatomies ...
Maria, Thom, Edward H, Bertram
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In the last two decades our understanding of MTLE and its pathophysiology has grown remarkably. Perhaps the most important recognition is that it is not a single entity with a uniform pathology. Rather, it is associated with significant variations in pathology that, in turn, are likely associated with different causes, functional anatomies ...
Maria, Thom, Edward H, Bertram
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Neocortical Temporal Lobe Epilepsy
Journal of Clinical Neurophysiology, 2012Neocortical temporal lobe epilepsy (NTLE) comprises a heterogeneous group of epilepsies with focal seizures characterized by auditory, somatosensory, or psychic auras followed by motionless staring, early contralateral clonic activity often secondarily generalizing.
Jeffrey D, Kennedy, Stephan U, Schuele
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Networks in Temporal Lobe Epilepsy
Neurosurgery Clinics of North America, 2020Temporal lobe epilepsy (TLE) is the most common type of drug-resistant focal epilepsy. Epilepsy can be conceptualized as a network disorder with the epileptogenic zone a critical node of the network. Temporal lobe networks can be identified on the microscale and macroscale, both during the interictal and ictal periods.
Karina A, González Otárula +1 more
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Chromogranins in Temporal Lobe Epilepsy
Epilepsia, 2000Summary: Purpose: Chromogranins are neuropeptide precursors stored in large dense core vesicles. Because physiological functions have been postulated for peptides originating from chromogranins, we investigated the distribution of chromogranins A and B and secretoneurin (a peptide derived from secretogranin II) in the control and epileptic ...
S, Kandlhofer +6 more
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Genetics of temporal lobe epilepsy
Brain and Development, 2012The most common partial epilepsy, temporal lobe epilepsy (TLE) consists of a heterogeneous group of seizure disorders originating in the temporal lobe. TLE had been thought to develop as a result of acquired structural problems in the temporal lobe.
Su-Kyeong, Hwang, Shinichi, Hirose
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Hypersalivation in Temporal Lobe Epilepsy
Epilepsia, 2006Summary: Purpose: We sought to determine whether hypersalivation helps lateralize seizure onset during complex partial seizures of temporal lobe origin. Several clinical signs, which help lateralize seizure onset, have been reported in temporal lobe epilepsy (TLE). Increased salivation only occasionally has been reported as a manifestation of partial
Jagdish, Shah +3 more
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Violence and temporal lobe epilepsy
American Journal of Psychiatry, 1984Note: This publication comments on the article by O. Devinsky and D. Bear discussing the positive benefits of neurosurgical intervention to control violence in temporal lobe epilepsy. Devinsky, O., & Bear, D.M. (1984, May). Varieties of Aggressive Behavior in Temporal Lobe Epilepsy. American Journal of Psychiatry, 141(5), 651-656. (VioLit Record Number
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Anatomic Temporal Lobe Resections for Temporal Lobe Epilepsy
Neurosurgery Clinics of North America, 1993Temporal lobe epilepsy is not a single clinicopathologic entity but a group of syndromes requiring different surgical solutions. Anatomic resections planned for the treatment of these syndromes are aimed at pathologic substrates minimizing ablation of normal tissue. Most of these procedures involve mesial and lateral temporal resections.
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Temporal lobe epilepsy and endocrinopathy
Acta Neurovegetativa, 1964Following a previous publication of three cases with temporal lobe epilepsy and endocrinopathy, the authors report two more similar cases.
G, ANASTASOPOULOS, D, KOKKINI
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Introduction to temporal lobe epilepsy
Epilepsy Research, 1996Epileptic disorders are classified as idiopathic when they are genetically transmitted conditions that consist of epilepsy only, with no structural lesions in the brain and no associated neurological deficits, and symptomatic when they result from some other primary brain lesion or insult.
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