Results 311 to 320 of about 325,450 (339)
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Emergency Medicine Clinics of North America, 1993
Emergency physicians often must evaluate patients with thrombocytopenia. Once recognized, the initial concern is for evaluating the patient's hemostasis. Prompt diagnosis and early intervention may be life saving. This article reviews platelet function and kinetics and presents the pathophysiology and clinical and laboratory findings of a number of ...
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Emergency physicians often must evaluate patients with thrombocytopenia. Once recognized, the initial concern is for evaluating the patient's hemostasis. Prompt diagnosis and early intervention may be life saving. This article reviews platelet function and kinetics and presents the pathophysiology and clinical and laboratory findings of a number of ...
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Early Human Development, 2005
Thrombocytopenia occurs in up to a third of preterm neonates admitted to intensive care units. In these babies, thrombocytopenia typically presents in one of two patterns: early-onset thrombocytopenia occurring within 72 h of birth and late-onset thrombocytopenia which develops after 72 h.
Subarna, Chakravorty +2 more
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Thrombocytopenia occurs in up to a third of preterm neonates admitted to intensive care units. In these babies, thrombocytopenia typically presents in one of two patterns: early-onset thrombocytopenia occurring within 72 h of birth and late-onset thrombocytopenia which develops after 72 h.
Subarna, Chakravorty +2 more
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Australian and New Zealand Journal of Medicine, 1987
Abstract Lupoid thrombocytopenia is herein defined as a disorder in which patients with throm‐bocytopenia have a positive Hep 2 antinuclear antibody. Although these patients may have other clinical or laboratory findings consistent with systemic lupus erythematosus, they do not satisfy the revised diagnostic criteria set down by the American Rheumatism
B G, Firkin +3 more
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Abstract Lupoid thrombocytopenia is herein defined as a disorder in which patients with throm‐bocytopenia have a positive Hep 2 antinuclear antibody. Although these patients may have other clinical or laboratory findings consistent with systemic lupus erythematosus, they do not satisfy the revised diagnostic criteria set down by the American Rheumatism
B G, Firkin +3 more
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Clinics in Perinatology, 1984
In this well-detailed review, the authors review the contribution of platelets to hemostasis, diagnostic approaches to the thrombocytopenic infant, and the classification and treatment of neonatal thrombocytopenia.
M, Andrew, J, Kelton
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In this well-detailed review, the authors review the contribution of platelets to hemostasis, diagnostic approaches to the thrombocytopenic infant, and the classification and treatment of neonatal thrombocytopenia.
M, Andrew, J, Kelton
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Clinical Obstetrics and Gynecology, 1999
For the fetuses who are at risk for antenatal or postnatal sequelae from AIT, prevention and treatment are now possible. This requires the attention of the obstetrician to factors in the patient's history and early referral to a center experienced in the diagnosis and management of fetal AIT.
D W, Skupski, J B, Bussel
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For the fetuses who are at risk for antenatal or postnatal sequelae from AIT, prevention and treatment are now possible. This requires the attention of the obstetrician to factors in the patient's history and early referral to a center experienced in the diagnosis and management of fetal AIT.
D W, Skupski, J B, Bussel
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Clinics in Perinatology, 1995
Maternal thrombocytopenia is common in normal pregnancies, but is a poor predictor of fetal thrombocytopenia even when the maternal thrombocytopenia is of pathologic etiology. The real risk to the fetus and the neonate is alloimmune thrombocytopenia, although identification of index cases is problematic and management of future pregnancies has not been
R F, Burrows, J G, Kelton
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Maternal thrombocytopenia is common in normal pregnancies, but is a poor predictor of fetal thrombocytopenia even when the maternal thrombocytopenia is of pathologic etiology. The real risk to the fetus and the neonate is alloimmune thrombocytopenia, although identification of index cases is problematic and management of future pregnancies has not been
R F, Burrows, J G, Kelton
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Seminars in Fetal and Neonatal Medicine, 2008
Fetal thrombocytopenia is most often caused by maternal alloantibodies against fetal platelets crossing the placenta and resulting in platelet destruction. This condition, known as fetal and neonatal alloimmune thrombocytopenia, is usually detected after the birth of a symptomatic child who shows signs of bleeding in the skin or in the brain.
L, Porcelijn +2 more
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Fetal thrombocytopenia is most often caused by maternal alloantibodies against fetal platelets crossing the placenta and resulting in platelet destruction. This condition, known as fetal and neonatal alloimmune thrombocytopenia, is usually detected after the birth of a symptomatic child who shows signs of bleeding in the skin or in the brain.
L, Porcelijn +2 more
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Current Opinion in Hematology, 1995
Congenital thrombocytopenias are rare bleeding disorders but must be included in the differential diagnosis when investigating a young infant with chronic thrombocytopenia. Several of these syndromes have associated defects of immune, renal, or skeletal systems in addition to thrombocytopenia.
I, Warrier, J M, Lusher
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Congenital thrombocytopenias are rare bleeding disorders but must be included in the differential diagnosis when investigating a young infant with chronic thrombocytopenia. Several of these syndromes have associated defects of immune, renal, or skeletal systems in addition to thrombocytopenia.
I, Warrier, J M, Lusher
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Hospital Practice, 1983
The common denominator of this group of disorders is bleeding due to antibody-mediated platelet destruction. The variations in clinical manifestation--ranging from mild to fatal--reflect the diverse origins of the troublesome antibodies: transplacental, exogenous, and autogenous.
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The common denominator of this group of disorders is bleeding due to antibody-mediated platelet destruction. The variations in clinical manifestation--ranging from mild to fatal--reflect the diverse origins of the troublesome antibodies: transplacental, exogenous, and autogenous.
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Journal of Pediatric Hematology/Oncology, 2003
Alloimmunization against platelet antigens may take place during pregnancy and following transfusion or transplantation. The resulting antibodies bind to the target platelet antigens and may lead to a rapid sequestration of incompatible platelets.
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Alloimmunization against platelet antigens may take place during pregnancy and following transfusion or transplantation. The resulting antibodies bind to the target platelet antigens and may lead to a rapid sequestration of incompatible platelets.
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