Results 21 to 30 of about 2,081,679 (301)

Waterhouse friderichsen syndrome in a case of staphylococcal toxic shock syndrome

open access: yesJournal of Pediatric Critical Care, 2016
Staphylococcal toxic shock syndrome (TSS) is a rare and potentially fatal multi system dysfunction. The syndrome occurs primarily due to TSS Toxin-1 (TSS-1) liberated by Staphylococcus aureus (SA).
Devdeep Mukherjee   +2 more
doaj   +1 more source

Interaction of Staphylococcal Toxic Shock Syndrome Toxin-1 and Enterotoxin A on T Cell Proliferation and TNFα Secretion in Human Blood Mononuclear Cells

open access: yesCanadian Journal of Infectious Diseases, 1999
BACKGROUND: The majority of menstrual toxic shock syndrome (MTSS) cases are caused by a single clone of Staphylococcus aureus that produces both toxic shock syndrome toxin-1 (TSST-1) and staphylococcal enterotoxin A (SEA).
Monica L De Boer   +2 more
doaj   +1 more source

Síndrome do choque tóxico por Streptococcus pyogenes.

open access: yesActa Médica Portuguesa, 2011
Recently there has been an exponential increase in invasive infections caused by Streptococcus ß hemolyticus group A. In about one third of cases they are complicated by toxic shock syndrome, characterized by septic shock and multiorgan failure.
Rui Antunes   +4 more
doaj   +1 more source

Detection of Toxic Shock Syndrome Toxin-1 among Children Undergoing Surgical Operations

open access: yesمجلة كلية الطب, 2017
Background: Toxic-shock syndrome (TSS) is an acute onset; multiorgan disease caused mainly by Toxic-shock syndrome toxin-1 (TSST-1) producing Staphylococcus aureus strains.Testing for TSST-1 or anti-TSST-1 antibodies in the clinical setting may help to ...
Abdul-Razak Sh. Hasan   +2 more
doaj   +1 more source

Toxic shock syndrome

open access: yesPrimary Care Update for OB/GYNS, 2000
Toxic shock syndrome (TSS) is an acute febrile illness accompanied by watery diarrhea, sunburn rash, vomiting, hypotension, and multiple organ involvement. When it was initially recognized 20 years ago, TSS was primarily associated with menstruation. Since then, the frequency of menstrual TSS has decreased dramatically.
Department of Obstetrics and Gynecology, University of Florida College of Medicine, Gainesville, Florida, USA ( host institution )   +1 more
openaire   +3 more sources

Group B Streptococcal Toxic Shock Syndrome and covR/S Mutations Revisited

open access: yesEmerging Infectious Diseases, 2017
Gene mutations in the virulence regulator CovR/S of group A Streptococcus play a substantial role in the pathogenesis of streptococcal toxic shock syndrome.
Parham Sendi   +3 more
doaj   +1 more source

The Association Between Onset of Staphylococcal Non-menstrual Toxic Shock Syndrome With Inducibility of Toxic Shock Syndrome Toxin-1 Production

open access: yesFrontiers in Microbiology, 2022
Non-menstrual toxic shock syndrome (non-mTSS) is a life-threatening disease caused by Staphylococcus aureus strains producing superantigens, such as staphylococcal enterotoxins A, B, C, and toxic shock syndrome toxin-1 (TSST-1).
Yusuke Taki   +18 more
doaj   +1 more source

Streptococcal Toxic Shock Syndrome: Life Saving Role of Peritoneal Lavage and Drainage

open access: yes, 2016
Purpose We encountered a case where an infection with group A streptococcus (GAS; ie, Streptococcus pyogenes ) initially caused primary peritonitis and then subsequently caused streptococcal toxic shock syndrome. The patient's life was likely saved by an
Tomonori Kasai   +10 more
core   +1 more source

Streptococcal toxic shock syndrome in an adolescent

open access: yesМедицинский совет, 2022
A  special form of  streptococcal infection is streptococcal toxic shock syndrome  (STS), characterized by rapid development of symptoms and high mortality. Patient O., 14 years old, was taken to the infectious diseases department of OGAUZ DBNo. 1 by the
N. A. Ryzhakova   +8 more
doaj   +1 more source

Microbiome−host proteostasis crosstalk—An emerging perspective on mechanisms and interventions toward healthy longevity

open access: yesFEBS Letters, EarlyView.
Proteostasis and the gut microbiota play a key role in shaping host physiology. Microbiota‐derived metabolites, vitamins, and RNA modulate host proteostasis. Findings from model systems, including C. elegans, indicate microbes can either stabilize or disrupt host proteostasis.
Abhishek Anil Dubey, Maria Ermolaeva
wiley   +1 more source

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