Results 211 to 220 of about 3,820 (228)
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DNA fingerprinting reveals relationships between strains of Trypanosoma rangeli and Trypanosoma cruzi

1993
Very little is known about the structure and sequence of the genomic DNA and kDNA of T. rangeli and no highly polymorphic markers are known. In this paper, we show that the Jeffreys' multilocal probe 33.15 produces characteristic DNA fingerprints with these trypanosomes. The multiband patterns can be used to differentiate T. cruzi from T.
A M, Macedo   +3 more
openaire   +2 more sources

Differentiation of Trypanosoma cruzi and Trypanosoma rangeli of Colombia using minicircle hybridization tests

Diagnostic Microbiology and Infectious Disease, 2010
Although Trypanosoma rangeli is harmless for humans, it is a serious problem since it may be confused with diagnosis of Trypanosoma cruzi, the etiologic agent of Chagas disease. Both parasites overlap geographically, share antigenic protein, and are able to infect the same Triatominae vector and vertebrate host, including human.
Adriana, Botero   +4 more
openaire   +2 more sources

Use of Micro-Elisa for Quantitating Antibody to Trypanosoma Cruzi and Trypanosoma Rangeli

The American Journal of Tropical Medicine and Hygiene, 1979
Of 229 residents of a Panamanian village where both Trypanosoma cruzi and Trypanosoma rangeli are endemic, 52% had antibody to one or both species by the enzyme-linked immunosorbent assay (ELISA), 26% were positive by complement fixation, 17% were positive by direct agglutination, and 32% were positive on the basis of clinical impression.
R L, Anthony, C M, Johnson, O E, Sousa
openaire   +2 more sources

Vaccination with Trypanosoma rangeli induces resistance of guinea pigs to virulent Trypanosoma cruzi

Veterinary Immunology and Immunopathology, 2014
Chagas' disease, endemic in Latin America, is spread in natural environments through animal reservoirs, including marsupials, mice and guinea pigs. Farms breeding guinea pigs for food are located in some Latin-American countries with consequent risk of digestive infection.
B, Basso, E, Moretti, R, Fretes
openaire   +2 more sources

Trypanosoma rangeli: Effect on excretion in Rhodnius prolixus

Journal of Invertebrate Pathology, 1971
Abstract Excretion is severely reduced in Rhodnius prolixus infected with Trypanosoma rangeli , particularly in those bugs with hemocoelic infections. Several factors probably contribute to this reduction: tissue damage, lack of diuretic hormone or the presence of a chemical inhibitor in the hemolymph, and changes in its osmotic pressure.
openaire   +2 more sources

Histology of Rhodnius prolixus infected with Trypanosoma rangeli

Journal of Invertebrate Pathology, 1971
Abstract Intracellular stages of Trypanosoma rangeli develop in most tissues of infected Rhodnius prolixus , but they show a predilection for gut musculature, fat body, epidermis, and salivary glands. Heavy hemocoelic infections often result in severe nerve damage and hypertrophy of tracheal cells, which finally rupture and release flagellates. T.
openaire   +2 more sources

Comparative study of Trypanosoma rangeli and Trypanosoma cruzi telomeres

Molecular and Biochemical Parasitology, 2002
Miguel A, Chiurillo   +2 more
openaire   +2 more sources

Ultrastructural Morphology of Trypanosoma rangeli

Archiv für Protistenkunde, 1994
F. Tejero   +2 more
openaire   +1 more source

Growth characteristics of Trypanosoma rangeli

Transactions of the Royal Society of Tropical Medicine and Hygiene, 1976
E. Minter-Goedbloed   +1 more
openaire   +1 more source

Trypanosoma rangeli

1992
Antonio D’Alessandro-Bacigalupo   +1 more
openaire   +1 more source

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