Tumoral calcinosis in chronic renal failure: A case report and literature review. [PDF]
Andriandi, Husnul F, Tirta C.
europepmc +2 more sources
Ocular involvement in tumoral calcinosis
We report a 32-year-old male who presented with blurring of vision in the right eye since 1.5 years. He had history of swelling over the extensor surfaces of large joints which were migratory in nature.
Harsha Bhattacharjee +2 more
doaj +2 more sources
Hyperphosphatemic Tumoral Calcinosis With Pemigatinib Use. [PDF]
Puar A +6 more
europepmc +2 more sources
[Iclan's tumoral calcinosis in an elderly subject]. [PDF]
Cherkaoui GB, Oufkir AA.
europepmc +3 more sources
Uremic tumoral calcinosis with atypical manifestation. Case report
Arbey Aristizábal-Alzate +5 more
openalex +2 more sources
Tumoral calcinosis with pubic bone destruction: A case report
Haixiang Miao, Jihang Dai
doaj +2 more sources
Type 1 Hyperphosphatemic Familial Tumoral Calcinosis Associated With a Homozygous Variant Mutation in the GALNT3 Gene. [PDF]
Alghubishi SA +3 more
europepmc +2 more sources
Autoimmune hyperphosphatemic tumoral calcinosis in a patient with FGF23 autoantibodies [PDF]
Hyperphosphatemic familial tumoral calcinosis (HFTC)/hyperostosis-hyperphosphatemia syndrome (HHS) is an autosomal recessive disorder of ectopic calcification due to deficiency of or resistance to intact fibroblast growth factor 23 (iFGF23). Inactivating
Burbelo +14 more
core +1 more source
Tumoral Calcinosis in the Larynx
Tumoral calcinosis is a rare benign disease defined as calcium salt deposits in the periarticular soft tissue region. Tumoral calcinosis is rare in the neck and larynx.
Lianlian Liu +3 more
semanticscholar +1 more source
An Unusual Combination of Neurological Manifestations and Sudden Vision Loss in a Child with Familial Hyperphosphatemic Tumoral Calcinosis [PDF]
Hyperphosphatemia in the absence of renal failure is an unusual occurrence, particularly in children, but is a common primary feature of familial hyperphosphatemic tumor calcinosis.
Acton, Dena +7 more
core +1 more source

