Results 31 to 40 of about 17,609 (215)

Celiac disease in type 1 diabetes mellitus

open access: yesItalian Journal of Pediatrics, 2012
Celiac Disease (CD) occurs in patients with Type 1 Diabetes (T1D) ranging the prevalence of 4.4-11.1% versus 0.5% of the general population. The mechanism of association of these two diseases involves a shared genetic background: HLA genotype DR3-DQ2 and
Camarca Maria   +10 more
doaj   +1 more source

Liver Damage in Type 1 Diabetes Mellitus

open access: yesРоссийский журнал гастроэнтерологии, гепатологии, колопроктологии, 2021
Aim. An up-to-date review of the prevalence, pathogenesis, diagnosis and management of hepatological complications of type 1 diabetes mellitus (T1D).Key points. Diabetes type 1 causes a markedly more common liver injury than traditionally assumed.
A. O. Bueverov, A. V. Zilov
doaj   +1 more source

Vitamin D and Type 1 Diabetes Mellitus

open access: yesMìžnarodnij Endokrinologìčnij Žurnal, 2016
The article presents data concerning the problem of vitamin D deficiency in adolescents with diabetes mellitus type 1. A correlation between vitamin D deficiency and compensation of the disease is shown.
К.S. Biliaieva   +3 more
doaj   +1 more source

Cardiovascular risk in type 1 diabetes mellitus

open access: yesIndian Journal of Endocrinology and Metabolism, 2013
Commonly cardiovascular risk (CVR) is linked to type 2 diabetes mellitus as this type is known to be part of the metabolic syndrome, which includes other cardiovascular factors such as hypertension, dyslipidemia.
Ines Ben Hadj Slama Slim
doaj   +1 more source

Loeys-Dietz Syndrome with Type 1 Diabetes Mellitus

open access: yesClinical Pediatric Hematology-Oncology, 2023
Loeys-Dietz syndrome is a hereditary connective-tissue disorder first reported in 2005. It is caused by genetic defects in the transforming growth factor-beta (TGF-β) signaling pathway.
Daro Jeong, Jung Hyun Lee, Seom Gim Kong
doaj   +1 more source

Enteroviruses and type 1 diabetes mellitus

open access: yesBiomedicine & Pharmacotherapy, 2003
Despite decades of research, the etiology of type 1 diabetes mellitus (DM) is unknown. Several risk factors have been associated with type 1 DM, including viral infections, genetic predisposition, nutritional factors, and chemicals. Several investigators hypothesize that the etiologies of type 1 DM result from a complex interaction of genetic and ...
Harry W, Haverkos   +3 more
openaire   +2 more sources

Bone remodeling in type 1 diabetes mellitus

open access: yesБюллетень сибирской медицины, 2018
The aim of the study is to assess the effect of changes in the body in men and women with type 1 diabetes on the state of bone mineral density and metabolic rate and determine the directionality of changes in serum markers of bone remodeling and bone ...
S. S. Safarova
doaj   +1 more source

Identification of Micronuclei in the Lymphocytes of the Type 2 Diabetes Mellitus According to the Period of Diagnosis as a Biomarker

open access: yesPolytechnic Journal, 2023
Micronuclei in lymphocytes can indicate DNA damage or chromosomal instability. This study aimed to compare the Micronuclei to the different period diagnoses of type 2 diabetes mellitus. Four groups were created out of 140 people: type 2 diabetes mellitus
Harem Othman Smail
doaj   +1 more source

The Impact of Tenapanor on Body Composition and Blood Pressure in Peritoneal Dialysis Patients: A Pilot Study

open access: yesTherapeutic Apheresis and Dialysis, EarlyView.
ABSTRACT Instruction Tenapanor potentially affects fluid volume and blood pressure. This study explored its effects on these parameters in patients undergoing peritoneal dialysis (PD). Methods We conducted a single‐arm, prospective observational study that enrolled 10 patients on PD with hyperphosphatemia who were newly initiated on tenapanor.
Shun Yoshida   +11 more
wiley   +1 more source

Microbiome‐blood–brain barrier interactions in aging — mechanisms and therapeutic potential

open access: yesFEBS Letters, EarlyView.
Aging reshapes the gut microbiome (↓SCFA‐producing commensals; ↑pro‐inflammatory outputs), shifting circulating metabolites (↓SCFAs; ↑LPS, ↑TMAO, ↑PAA) that act at the BBB to increase nonspecific transcytosis, alter transport, and promote astrocyte reactivity, heightening brain vulnerability.
Daniel Cuervo‐Zanatta   +3 more
wiley   +1 more source

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