Results 141 to 150 of about 142,290 (306)

Proportions of underweight over time in Sub Saharan Africa.

, 2014
Proportions of underweight over time in Sub Saharan Africa.
Stella K. Muthuri (543854), Allana G. LeBlanc (422298), Mark S. Tremblay (127037), Claire E. Francis (543855), Lucy-Joy M. Wachira (543856), Margaret Sampson (68417), Vincent O. Onywera (543857)   +6 more
core   +1 more source

Wasting coexisting with underweight and stunting among children aged 6‒59 months hospitalised in Garissa County Referral Hospital, Kenya

Maternal and Child Nutrition
Management of undernourished children depends only on wasting yet it can coexist with underweight and/or stunting. Among children admitted to hospital with acute illness, we determined the proportion with wasting coexisting with underweight and/or ...
Mutuvi Wambua, Symon M. Kariuki, Hassan Abdullahi, Osman A. Abdullahi, Moses M. Ngari   +4 more
doaj   +1 more source

Factors influencing concurrent wasting, stunting, and underweight among children under five who suffered from severe acute malnutrition in low- and middle-income countries: a systematic review

Frontiers in Nutrition
BackgroundWasting, stunting, and underweight in children are complex health challenges shaped by a combination of immediate, underlying, and systemic factors.
Godana Arero Dassie, Tesfaye Chala Fantaye, Tesfaye Getachew Charkos, Midhakso Sento Erba, Fufa Balcha Tolosa   +4 more
doaj   +1 more source

Intrapatient tumour heterogeneity and clonal evolution in an autopsy study of metastatic salivary gland cancer

Molecular Oncology, EarlyView.
Tumour heterogeneity and clonal evolution of metastatic salivary gland cancer were evaluated in two patients with adenoid carcinoma and one patient with myoepithelial carcinoma. Radiology‐guided autopsy enabled multi‐region sampling (total samples n = 149), followed by whole‐genome sequencing and phylogenetic reconstruction (17 tumour samples, 4–7 per ...
Gerben Lassche, Niels J. van Ruitenbeek, Charlotte Adang, Luke O'Gorman, Pui Yuen Lee, Willemijn M. Klein, Adriana C. H. van Engen‐van Grunsven, Jack A. Schalken, Sander Bervoets, Gerald W. Verhaegh, Carla M. L. van Herpen   +10 more
wiley   +1 more source

Somatostatin receptor 4 (SSTR4) is a tumor suppressor in cutaneous and head & neck squamous cell carcinomas

Molecular Oncology, EarlyView.
This study identifies somatostatin receptor 4 (Sstr4) as a critical tumor suppressor against skin and head/neck cancers (HNSCC, cSCC, and BCC). The loss of Sstr4 removes a check on cell growth, causing hyperactivation of the MAPK‐ERK signaling pathway (↑).
Ali Taqvi, Tuan Khang Huynh, Adway Kadam, Zahra Kardan, Yue Yan, Amina Jamal Laham, Sampath Kumar Loganathan   +6 more
wiley   +1 more source

Wasting, underweight, and stunting by randomized treatment group.

Wasting, underweight, and stunting by randomized treatment group.
Valentin Boudo (8084525), Catherine E. Oldenburg (9379353), Mamadou Ouattara (128981), Clarisse Dah (10321042), Ali Sié (86539), Huiyu Hu (4381690), Thierry Ouedraogo (17833820), Mamadou Bountogo (8084519), Thomas M. Lietman (7252595), Kieran S. O’Brien (5166692), Benjamin F. Arnold (7255817), Elodie Lebas (5954207)   +11 more
core   +1 more source

Mechanisms and therapeutic opportunities of the ribotoxic stress response in cancer

Molecular Oncology, EarlyView.
Cancer cells' high translational demand creates opportunities to therapeutically target ribosome function. Ribosome stalling and collisions activate ZAKα and the ribotoxic stress response (RSR), which can trigger rapid, p53‐independent apoptosis in cancer.
Anastassiya Kim, Maryam Aslam, Elouise Torruella, Alphons Sony, Asif Rahman, Simira Smith, Columba de la Parra, Moira Sauane   +7 more
wiley   +1 more source

Regulation of the lncRNA NEAT1 by p53‐ΔNp63 crosstalk modulates the DNA damage response and therapeutic efficacy in HNSCC

Molecular Oncology, EarlyView.
In head and neck squamous cell carcinoma (HNSCC) p53 and p63 exert opposite roles on the transcription regulation of the lncRNA NEAT1. Under basal conditions, p53 levels are low and p63 represses NEAT1 expression. Upon genotoxic stress, p53 is rapidly induced, displacing p63 from the NEAT1 promoter leading to NEAT1 transcriptional activation and ...
Sara De Domenico, Veronica La Banca, Silvia D'Amico, Stefano Scalera, Sara Nicolai, Angelo Peschiaroli   +5 more
wiley   +1 more source

BODY IMAGE, SIKAP DAN PENGETAHUAN GIZI REMAJA PUTRI PADA UNDERWEIGHT DI MTS AL-WATHONIYYAH SEMARANG

, 2019
Latar Belakang : Prevalensi underweight pada remaja usia 13-15 tahun di Indonesia menurut Riskesdas 2013 sebanyak 11,1% sedangkan pada provinsi Jawa Tengah sebesar 11,4%.
FIRMANTI NAFIA MUGHNI SANDI
core  

APOBEC3 activity and DNA polymerase‐ε deficiency are associated with distinct IDH1 R132 hotspot mutations

Molecular Oncology, EarlyView.
Isocitrate dehydrogenase 1 (IDH1) mutations are highly recurrent in multiple human cancer types, including cholangiocarcinoma and glioma. IDH1 R132C is the most common IDH1 mutation in cholangiocarcinoma and likely arises from APOBEC3A‐ or APOBEC3B‐mediated deamination.
Kelly E. Butler, Bilal A. Lone, Ecem Unal, A. Rouf Banday   +3 more
wiley   +1 more source