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Vasopressin and vasopressin antagonists in heart failure and hyponatremia
Current Heart Failure Reports, 2008Increased synthesis of arginine vasopressin (AVP) plays a critical role in fluid retention and hyponatremia in patients with heart failure. The AVP receptor antagonists constitute a new class of agents that are promising in the management of hyponatremia and congestion.
Dimitrios, Farmakis +3 more
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Renal microvascular effects of vasopressin and vasopressin antagonists
American Journal of Physiology-Renal Physiology, 1989The effects of vasopressin (AVP) and vasopressin antagonists on lumen diameters of cortical afferent and efferent arterioles isolated from rabbit kidneys were examined. Over a concentration range of 10(-14) to 10(-7) M, AVP had no effect on lumen diameters of afferent arterioles, although the arterioles were responsive to norepinephrine.
R M, Edwards, W, Trizna, L B, Kinter
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Vasopressin and Vasopressin Receptor Antagonists in Heart Failure
Cardiology in Review, 2009Antidiuretic hormone, also known as arginine vasopressin, is a hormone with a multitude of physiologic activities including the control of urinary free water excretion. Antidiuretic hormone also plays a role in vasoconstriction and has 3 receptors that have been identified.
Gerard, Oghlakian, Marc, Klapholz
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Vasopressin metabolites: A link between vasopressin and memory?
1999The effects of endogenous metabolites of the neuropeptide vasopressin (VP) in behavioural tests led to the hypothesis that VP metabolites have a more selective function than VP. In contrast to VP, no peripheral effects have been found thus far with VP metabolites and their function seems to be associated with memory-related behaviour.
L G, Reijmers +4 more
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Vasopressin in Cardiovascular Control: Role of Circulating Vasopressin
Clinical Science, 1984Vasopressin has been shown to elicit vasoconstriction in unanaesthetized animals at plasma concentrations similar to those associated with its renal antidiuretic effect. The vasconstrictor effects of vasopressin do not normally translate into pressor responses until relatively high plasma concentrations are reached.
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