Results 181 to 190 of about 137,225 (329)

Venous Insufficiency: Endovascular and Surgical Treatment. [PDF]

open access: yesCurr Cardiol Rep
Khan A   +4 more
europepmc   +1 more source

Human Atlas of Tooth Decay Progression: Identification of Cellular Mechanisms Driving the Switch from Dental Pulp Repair Toward Irreversible Pulpitis

open access: yesAdvanced Science, EarlyView.
Tooth decay progression transforms the dental pulp response from repair to fibrosis. At early stages, stromal cells reprogram to repair the extra cellular matrix (ECM), blood vessels, and nerves, remodel and grow, keeping repair possible. In advanced decay, hypoxia, and vessel regression, in complement with an immune switch, fuel nerve degeneration and
Hoang Thai Ha   +12 more
wiley   +1 more source

Systematic review and meta-analysis of the impact of diabetes mellitus on chronic venous insufficiency. [PDF]

open access: yesJ Vasc Bras
de Castro ACF   +5 more
europepmc   +1 more source

Engineering Dimensional Configuration of Single‐Atom S‐Cu‐S Sites as Reversible Electron Station for Enhanced Peroxidase‐Mimicking

open access: yesAdvanced Science, EarlyView.
L‐cysteine triggers auto‐assembly of POD‐like 3D biomimetic S‐Cu‐S single‐atom nanozymes on MoS2 (MoCC). MoCC shows 16.3‐fold higher catalytic velocity and 17.9‐fold greater affinity than HRP, enabling efficient •OH generation via enhanced electron inversion and transfer.
Wenjie Ma   +12 more
wiley   +1 more source

Rapid post exercise venous refilling time is an independent contributor to chronic venous insufficiency. [PDF]

open access: yesJ Vasc Surg Venous Lymphat Disord
Elbenawi H   +6 more
europepmc   +1 more source

Chronic venous insufficiency and venous leg ulcers: Aetiology, on the pathophysiology-based treatment. [PDF]

open access: yesInt Wound J, 2023
Krizanova O   +5 more
europepmc   +1 more source

HRS Degradation‐Induced Nicotinamide Deficiency in Placental Extracellular Vesicles Triggers Preeclampsia by Disrupting Maternal‐Fetal Immune Homeostasis

open access: yesAdvanced Science, EarlyView.
This study shows that lower NAM levels in PE‐derived pEVs correlate with disease severity. NAM‐deficient pEVs reduce Th1 and Th17 inhibition, leading to PE‐like symptoms. NAM in pEVs inhibits Th1 via SIRT1 and Th17 via macrophages. Reduced NAM in PE‐EVs is due to decreased HRS expression in trophoblasts, resulting from elevated HSP27.
Haiyi Fei   +10 more
wiley   +1 more source

Patient Race and Ethnicity Do Not Predict Ulceration Among Ambulatory Patients With Venous Insufficiency. [PDF]

open access: yesWound Repair Regen
Nicholas A   +5 more
europepmc   +1 more source

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