Results 81 to 90 of about 179,560 (211)

CLINICAL PREDICTIVE VALUE OF TISSUE INHIBITOR OF METALLOPROTEINASE-1 IMYOCARDIAL REMODELING DEVELOPMENT IN PATIENTS HAVING Q-WAVE MYOCARDIAL INFARCTION

Атеросклероз, 2019
In this paper we thought to investigate clinical value of tissue inhibitor of metalloproteinase-1 (TIMP-1) in predicting left ventricular (LV) myocardial remodeling in patients having Q-wave myocardial infarction (Q-wave MI).
A. S. Vorobyev, L. V. Kovalenko, K. Yu. Nikolaev, I. A. Urvantseva, T. О. Simonyan, М. I. Rudenko   +5 more
doaj   +1 more source

Rabbit models of heart disease. [PDF]

, 2008
Human heart disease is a major cause of death and disability. A variety of animal models of cardiac disease have been developed to better understand the etiology, cellular and molecular mechanisms of cardiac dysfunction and novel therapeutic strategies ...
Bers, Donald M, Pogwizd, Steven M
core  

Gender differences in cardiac left ventricular mass and function: clinical and experimental observations [PDF]

, 2014
Background: The aim of this study was to evaluate gender-associated impact on left ventricular mass (LVM) and on left ventricular function (LVF) in humans and rats with aging. Methods: Myocyte area and collagen volume fraction (CVF) were studied in rats.
Aiello, Ernesto Alejandro, Diaz, Alejandro, Ennis, Irene Lucia, Escudero, Eduardo Manuel, Orlowski, Alejandro, Pinilla, Oscar Andres   +5 more
core   +2 more sources

The effect of rosuvastatin on inflammation, matrix turnover and left ventricular remodeling in dilated cardiomyopathy: a randomized, controlled trial.

PLoS ONE, 2014
BackgroundDilated cardiomyopathy is characterized by left ventricular dilatation and dysfunction. Inflammation and adverse remodeling of the extracellular matrix may be involved in the pathogenesis.
Kaspar Broch, Erik T Askevold, Erik Gjertsen, Thor Ueland, Arne Yndestad, Kristin Godang, Wenche Stueflotten, Johanna Andreassen, Rolf Svendsmark, Hans-Jørgen Smith, Svend Aakhus, Pål Aukrust, Lars Gullestad   +12 more
doaj   +1 more source

Hepatocyte growth factor gene therapy reduces ventricular arrhythmia in animal models of myocardial ischemia. [PDF]

, 2005
It was recently reported that gene therapy using hepatocyte growth factor (HGF) has the potential to preserve cardiac function after myocardial ischemia. We speculated that this HGF gene therapy could also prevent ventricular arrhythmia.
Aoki, Motokuni, Hashimoto, Katsushi, Kaneda, Yasufumi, Kusano, Kengo Fukushima, Morishita, Ryuichi, Nakamura, Kazufumi, Ohe, Tohru, Yumoto, Akihisa   +7 more
core   +1 more source

Right ventricular remodeling [PDF]

The Journal of Thoracic and Cardiovascular Surgery, 2006
Anderson, Robert H., Lunkenheimer, Paul P.   +1 more
openaire   +2 more sources

The impact of dose of the angiotensin-receptor blocker valsartan on the post-myocardial infarction ventricular remodeling: study protocol for a randomized controlled trial

Trials, 2011
Background Angiotensin-converting enzyme inhibitors and the angiotensin-receptor blocker valsartan ameliorate ventricular remodeling after myocardial infarction (MI).
Cho Young-Rak, Kim Young-Dae, Park Tae-Ho, Park Kyungil, Park Jong-Sung, Baek Heekyung, Choi Sun-Young, Kim Kee-Sik, Hong Taek-Jong, Yang Tae-Hyun, Hwang Jin-Yong, Park Jong-Seon, Hur Seung-Ho, Lee Sang-Gon   +13 more
doaj   +1 more source

Atrial cellular electrophysiological changes in patients with ventricular dysfunction may predispose to AF [PDF]

, 2009
<b>Background:</b> Left ventricular systolic dysfunction (LVSD) is a risk factor for atrial fibrillation (AF), but the atrial cellular electrophysiological mechanisms in humans are unclear.
Kane, K.A., Marshall, G.E., Norrie, J., Pau, D., Rankin, A.C., Redpath, C., Russell, J.A., Workman, A.J.   +7 more
core   +2 more sources

Cardiac hypertrophy and heart failure: from the case to review of literature [PDF]

, 2016
In response to an increased workload due to physiological or pathological stimuli, the heart may undergo a process of growth with increased muscle mass called cardiac hypertrophy. It is a particular mechanism of long term compensation used by the heart
Massoni, Francesco, Onofri, Emanuela, Ricci, Lidia, Ricci, Serafino, Simeone, Claudio   +4 more
core   +1 more source

Pathophysiological Basis of Right Ventricular Remodeling

Journal of Cardiovascular Pharmacology and Therapeutics, 2007
The pathophysiology of right ventricular (RV) remodeling is a complex process and may include unique elements not observed in left ventricular (LV) remodeling. The RV also has a relatively irregular geometry not accounted for in LV analyses. RV remodeling includes basic changes in geometry, wall thickness, and ventricular pressure-volume relationships.
Marcus, Kret, Rohit, Arora
openaire   +2 more sources