Results 131 to 140 of about 50,473 (244)

Complex Cyanotic Congenital Heart Disease in Pregnancy: Survival With Unrepaired Pulmonary Atresia and Ventricular Septal Defect - A Case Report. [PDF]

Clin Med Insights Cardiol
Muhali SS, Marcel L, Peter SC, Lwiza AF, Sadiq AM, Sadiq AM.   +5 more
europepmc   +1 more source

Unveiling New Insights: Reinterpreting DES Mutation, p.Arg383His, Through a Study of an Iranian Family With Isolated Hypertrophic Cardiomyopathy, Implication for Phenotype–Genotype Correlation Analysis

Clinical Case Reports, Volume 14, Issue 8, August 2026.
ABSTRACT Desmin, a crucial intermediate filament in muscle cells, maintains structural integrity in cardiac muscle and provides stability to striated muscle cells. Mutations in the DES gene lead to desminopathies, causing diverse cardiac and skeletal myopathies.
Saeideh Kavousi, Farzad Kamali, Bahareh Rabbani, Mehrdad Behmanesh, Nejat Mahdieh, Mehrdad Noruzinia   +5 more
wiley   +1 more source

Battling Right Ventricular Dysfunction in Post-Infarction Ventricular Septal Defect-A Case Report and Comprehensive Review of Literature. [PDF]

Life (Basel)
Moldovan H, Dobra I, Safta S, Robu M, Guta A, Preda S, Voicu A, Girel M, Alexandrescu A, Zaharia O.   +9 more
europepmc   +1 more source

Simultaneous Heart–Kidney Transplantation for Ebstein's Anomaly: A Viable Treatment Option in Latin America

Clinical Case Reports, Volume 14, Issue 8, August 2026.
Chest x‐ray showing a typical presentation of Ebstein anomaly with severe cardiomegaly and a prominent right atrium silhouette. ABSTRACT Simultaneous heart–kidney transplantation is a viable and effective treatment option for patients with congenital heart disease and advanced chronic kidney disease when both conditions threaten survival.
Edgar García‐Cruz, Andrea Luna‐Hernández, Jesús Alberto Blanco‐Hernández, Jorge Sánchez‐Nieto, Stephanie Teresa Angulo‐Cruzado, Karla Alejandra Pupiales‐Dávila, Carla Domínguez‐Pereyra, Reyna Moreno‐Ruíz, Carlos Antonio Villegas‐Chávez, Antonio Jordán‐Ríos, Daniel Manzur‐Sandoval, Victor Alejandro Quiroz‐Martínez, Francisco Martín Baranda‐Tovar   +12 more
wiley   +1 more source

Atrioventricular Conduction Disturbance After Perimembranous Ventricular Septal Defect Closure. [PDF]

JACC Case Rep
Kwok GHT, Wong CK, Chen HR, Un KC.
europepmc   +1 more source

Lung Pericytes: Molecular Mechanisms, Signaling Pathways, and Roles in Pulmonary Diseases

Comprehensive Physiology, Volume 16, Issue 4, August 2026.
Central role of pericytes across major pulmonary diseases. Conceptual wheel diagram illustrating the central role of lung pericytes across diverse pulmonary disease states. A pericyte positioned at the center expresses canonical markers, including PDGFRβ, CD146, and NG2, and connects radially to five major disease contexts.
Stuti Agarwal, Anuradha Bankar, Vinicio A. de Jesus Perez   +2 more
wiley   +1 more source

Surgical repair of a ventricular septal defect following low-velocity blunt chest trauma. [PDF]

BMC Cardiovasc Disord
Yang Y, Zhou X, Tang H, Wei X.
europepmc   +1 more source

Timing of conception after methotrexate and subsequent pregnancy outcomes: A retrospective cohort study

International Journal of Gynecology &Obstetrics, Volume 174, Issue 2, Page 1061-1069, August 2026.
Abstract Objective Methotrexate is widely used to treat ectopic pregnancy (EP). Concerns about its teratogenicity have led to uncertainty regarding the optimal interval before attempting conception. Guidelines recommend a 3‐month washout period, while the manufacturer advises 6 months. Evidence on the safety of early conception is limited.
Bar Rosh, Joul Hadad, Naomi Gronich
wiley   +1 more source

Case Report: Endothelialized but unstable: late migration of a ventricular septal defect closure device. [PDF]

Front Cardiovasc Med
Jan MJ, Wang CC, Jan SL, Liu WL.
europepmc   +1 more source

A Mechanistic Review of Environmental Stressor to Decode Their Effect on Congenital Malformations: A Developmental Toxicity

Journal of Biochemical and Molecular Toxicology, Volume 40, Issue 8, August 2026.
Environmental toxicants may disrupt developmental pathways via strong molecular docking interactions with hub FGF proteins (FGF9 and FGF4) like dibenzo‐p‐dioxin (−7.2 kcal/mol) at the ASN146 residue of FGF9, driving congenital malformations as revealed by PPI networks and toxicity profiling.
Adarsh Kumar Shukla, Rashmi Mahajan, Anuj Kumar Tyagi, Kamendra Swaroop   +3 more
wiley   +1 more source