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Unruptured congenital sinus of Valsalva aneurysm causing right ventricular outflow tract obstruction associated with hidden ventricular septal defect: a case report. [PDF]
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Circulation, 1965
Records of 273 cases of ventricular septal defect from five participating institutions were studied. Insignificant hemodynamic disturbance (Roger's) was found in 11 per cent, moderate to large left-to-right shunts in 55 per cent, and severe pulmonary vascular disease ("Eisenmenger's") in 8 per cent.
D G, Ritter +3 more
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Records of 273 cases of ventricular septal defect from five participating institutions were studied. Insignificant hemodynamic disturbance (Roger's) was found in 11 per cent, moderate to large left-to-right shunts in 55 per cent, and severe pulmonary vascular disease ("Eisenmenger's") in 8 per cent.
D G, Ritter +3 more
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The Lancet, 2011
Ventricular septal defects account for up to 40% of all congenital cardiac malformations. The diagnosis encompasses a broad range of anomalies, including isolated defects and those associated with other congenital cardiac malformations. Presentation, symptoms, natural history, and management of ventricular septal defects depend on size and anatomical ...
Daniel J, Penny, G Wesley, Vick
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Ventricular septal defects account for up to 40% of all congenital cardiac malformations. The diagnosis encompasses a broad range of anomalies, including isolated defects and those associated with other congenital cardiac malformations. Presentation, symptoms, natural history, and management of ventricular septal defects depend on size and anatomical ...
Daniel J, Penny, G Wesley, Vick
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Multiple Ventricular Septal Defects
The Annals of Thoracic Surgery, 1972Abstract Multiple ventricular septal defects (VSD) were closed in 24 children aged 3 to 13 years (mean 6.7). The number of defects ranged from 2 to 7 (mean 4). There were 3 hospital deaths and 1 late death. Three patients required reoperation to close defects which had reopened or had been overlooked previously, and 1 of these died (the late death in ...
I M, Breckenridge +3 more
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Traumatic Ventricular Septal Defect
The Annals of Thoracic Surgery, 1967THE treatment of traumatic ventricular septal defects produced by penetrating injuries of the heart is a subject of controversy.
R L, Carter, H M, Albert, B A, Glass
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TREATMENT OF VENTRICULAR SEPTAL DEFECTS
Journal of the American Medical Association, 1957Experience with congenital defects of the interventricular septum has shown that the prognosis is poor if the defect and associated shunt flow are large, both ventricles are enlarged, and the pressures in right ventricle and pulmonary artery are elevated. The pressures found by catheterization are an important criterion of operability.
E B, KAY +3 more
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2014
The defects that may be suitable for percutaneous closure are located within the muscular septum (muscular ventricular septal defects, MVSD) or in the perimembranous septum (perimembranous ventricular septal defects, PVSD) with or without aneurysm, and they can be native of residual post surgery.
Chessa M., Butera G.
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The defects that may be suitable for percutaneous closure are located within the muscular septum (muscular ventricular septal defects, MVSD) or in the perimembranous septum (perimembranous ventricular septal defects, PVSD) with or without aneurysm, and they can be native of residual post surgery.
Chessa M., Butera G.
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Ventricular septal defects in the horse
Journal of the American Veterinary Medical Association, 1983Summary Clinical, echocardiographic, and right-side cardiac catheterization data were collected in 6 horses with ventricular septal defects. The defects were confirmed by necropsy in 5 horses. On echocardiography, the cardiac dimensions were normal in 3 horses and enlarged in 2 others. A step-up in partial oxygen pressure between right atrium and right
C W, Lombard +2 more
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Hydrodynamics in ventricular septal defects
American Heart Journal, 1956Abstract The hydraulics responsible for shunt flow in ventricular septal defects (VSD) were studied in a specially designed model which permitted adjustment of the size of the defect, changes in the volume of circulating fluid, and changes in the resistances to inflow and outflow of the several heart chambers.
P, BROSTOFF, S, RODBARD
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