Results 41 to 50 of about 52,903 (224)

The Impact of Specific Viruses on Clinical Outcome in Children Presenting with Acute Heart Failure [PDF]

open access: yes, 2016
: The presence and type of viral genomes have been suggested as the main etiology for inflammatory dilated cardiomyopathy. Information on the clinical implication of this finding in a large population of children is lacking.
Bassano, C   +7 more
core   +2 more sources

Roles of Host Immunity in Viral Myocarditis and Dilated Cardiomyopathy

open access: yesJournal of Immunology Research, 2018
The pathogenesis of viral myocarditis includes both the direct damage mediated by viral infection and the indirect lesion resulted from host immune responses.
Lifang Zhao, Zhaoying Fu
doaj   +1 more source

A20 (TNFAIP3) alleviates CVB3-induced myocarditis via inhibiting NF-κB signaling.

open access: yesPLoS ONE, 2012
BackgroundViral myocarditis, which is most prevalently caused by Coxsackievirus B3 (CVB3) infection, is a serious clinical condition characterized by cardiac inflammation.
Jun Gui   +4 more
doaj   +1 more source

The role of miRNAs in viral myocarditis, and its possible implication in induction of mRNA-based COVID-19 vaccines-induced myocarditis

open access: yesBulletin of the National Research Centre, 2022
Background Several reports of unheeded complications secondary to the current mass international rollout of SARS-COV-2 vaccines, one of which is myocarditis occurring with the FDA fully approved vaccine, Pfizer, and others.
Antoine AbdelMassih   +46 more
doaj   +1 more source

Toll-Like Receptors: Are They Taking a Toll on the Heart in Viral Myocarditis?

open access: yesViruses, 2021
Myocarditis is an inflammatory disease of the heart with viral infections being the most common aetiology. Its complex biology remains poorly understood and its clinical management is one of the most challenging in the field of cardiology.
Kasper Favere   +8 more
doaj   +1 more source

Microfluidic Modeling of Macrophage‐Induced Cardiac Inflammation Using NF‐κB Reporter Cardiomyocytes

open access: yesAdvanced Healthcare Materials, EarlyView.
A cardiac inflammation model was developed to enable real‐time analysis of macrophage‐cardiomyocyte interactions using NF‐κB‐reporter cardiomyocytes and a microfluidic co‐culture system. A MATLAB‐based model was implemented to optimize cell density and medium resupply based on TNFα diffusion kinetics. Activation thresholds and stress responses in NF‐κB‐
Chao Liu   +6 more
wiley   +1 more source

Acute Viral Hepatitis E Is Associated with the Development of Myocarditis

open access: yesCase Reports in Hepatology, 2015
Myocarditis, an inflammatory disease of heart muscle, is an important cause of dilated cardiomyopathy worldwide. Viral infection is an important cause of myocarditis. This condition presents with various symptoms, ranging from minimally symptomatic cases
M. Premkumar   +5 more
doaj   +1 more source

Viral myocarditis: etiology and pathogenesis, problems of diagnostics

open access: yesЖурнал инфектологии, 2014
The review provides data on the most important etiological factors of viral myocarditis and on relatively new pathogens, based on the capabilities of modern verification of viral diseases.
T. V. Antonova, N. S. Zhevnerova
doaj   +1 more source

Rhinovirus as a rare cause of acute onset dilated cardiomyopathy due to myocarditis in a newborn: case report and review of the literature

open access: yesThe Turkish Journal of Pediatrics, 2022
Background. Cardiomyopathies account for 1% of cardiac diseases that mainly originate from myocarditis in the form of dilated cardiomyopathy in the neonatal period.
Burcu Cebeci   +4 more
doaj   +1 more source

Proteasomal protein degradation: adaptation of cellular proteolysis with impact on virus-and cytokine-mediated damage of heart tissue during myocarditis [PDF]

open access: yes, 2018
Viral myocarditis is an inflammation of the heart muscle triggered by direct virus-induced cytolysis and immune response mechanisms with most severe consequences during early childhood.
Agarwal   +175 more
core   +1 more source

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