Results 171 to 180 of about 185,950 (370)

Von Willebrand disease [PDF]

Clinical Medicine, 2007
openaire   +3 more sources

Clinical practice guidelines for the management of atypical haemolytic uraemic syndrome in the United Kingdom [PDF]

, 2010
,, Goodship, Tim H J, Machin, Sam, Taylor, C Mark, Wigmore, Stephen J   +4 more
core   +1 more source

Determination of Fibrinogen Ratio Cutoff Limits Using Indirect Reference Interval Methodology

International Journal of Laboratory Hematology, EarlyView.
ABSTRACT Introduction Discordant fibrinogen antigen to activity ratios are utilized by clinicians as evidence of dysfibrinogenemia. Abnormal ratio cutoffs implemented by clinical laboratories are typically determined by validation studies that include limited numbers of samples.
Abdulrahman Saadalla, Kelly Doyle, Karen Moser, Kristi Smock   +3 more
wiley   +1 more source

Cosegregation of von Willebrand factor gene polymorphisms and possible germinal mosaicism in type IIB von Willebrand disease [PDF]

, 1991
Elizabeth W. Murray, AR Giles, PJ Bridge, IR Peake, D Lillicrap   +4 more
openalex   +1 more source

Common Hematologic Emergencies—Acute Promyelocytic Leukemia and Microangiopathic Hemolytic Anemias—A Pivotal Role of Clinical Laboratory

International Journal of Laboratory Hematology, EarlyView.
ABSTRACT Hematologic emergencies are urgent health conditions which result in significant mortality and morbidity unless timely therapeutic measures are taken. Therapeutic success depends on their timely and accurate recognition by hematology laboratory services.
Ganna Shestakova, Nicolas Ulrich Edgar, Anton V. Rets   +2 more
wiley   +1 more source

PHENOTYPIC STUDY AND MOLECULA ANALYSIS OF VON WILLEBRAND TYPE 1 PATIENTS

Majallah-i Dānishgāh-i ̒Ulūm-i Pizishkī-i Bābul, 2006
BACKGROUND & OBJECTIVE: Von willebrand disease (VWD) is the most common bleeding disorder caused by von willbrand factor (VWF) deficiency and autosomal dominance inheritance pattern. It is divided into three types, one and three (quantitative) and type 2
SMB Hashemi Soteh, N Rezaei, A Goodeve
doaj  

The molecular defect in type IIB von Willebrand disease. Identification of four potential missense mutations within the putative GpIb binding domain. [PDF]

, 1991
Kathleen A. Cooney, William C. Nichols, M E Bruck, Wadie F. Bahou, Amy D. Shapiro, Ejw Bowie, H R Gralnick, David Ginsburg   +7 more
openalex   +1 more source

Update on the pathophysiology and classification of von Willebrand disease: a report of the Subcommittee on von Willebrand Factor

Journal of Thrombosis and Haemostasis, 2006
J. Sadler, U. Budde, J. Eikenboom, E. Favaloro, F. Hill, L. Holmberg, J. Ingerslev, Christine A. Lee, D. Lillicrap, P. Mannucci, C. Mazurier, D. Meyer, W. Nichols, M. Nishino, I. Peake, F. Rodeghiero, R. Schneppenheim, Z. Ruggeri, A. Srivastava, R. Montgomery, A. Federici   +20 more
semanticscholar   +1 more source

Diagnostic Performance of Prothrombin Time and Activated Partial Thromboplastin Time in Children: A Service Evaluation

International Journal of Laboratory Hematology, EarlyView.
ABSTRACT Background Coagulation screening, consisting of prothrombin time (PT) and activated partial prothrombin time (aPTT), is routinely performed in paediatrics to identify bleeding disorders or guide peri‐procedural management. We evaluated the trends in utilisation and diagnostic yield of PT and aPTT testing as part of coagulation screening in a ...
Gerard Gurumurthy, Mikias Lemma, Lianna Reynolds, John Grainger, Jecko Thachil   +4 more
wiley   +1 more source

Molecular studies of von Willebrand disease: reduced von Willebrand factor biosynthesis, storage, and release in endothelial cells derived from patients with type I von Willebrand disease [published erratum appears in Blood 1990 Nov 1;76(9):1901] [PDF]

, 1990
Bruce M. Ewenstein, Aida Inbal, J S Pober, RI Handin   +3 more
openalex   +1 more source