Results 41 to 50 of about 76,947 (296)

A moment in time: from the digital record of a migrating library [PDF]

open access: yes, 2005
This paper draws on the work of the Arts & Humanities Research Council-funded Glasgow Cassirer Project, which makes correspondence and documents relating to the German philosopher Ernst Cassirer (1874-1945) available online. The paper concentrates on
Whitaker, G.
core  

Corrosion of bronzes by extended wetting with single versus mixed acidic pollutants [PDF]

open access: yes, 2014
The corrosion of bronzes was examined in the context of single-acid versus mixed-acid (as in urban acid rain) solutions. Two bi-component bronzes (copper with either 3% Sn or 7% Sn) that closely represent those of historic artifacts were immersed for ...
Adriaens, Mieke   +3 more
core   +2 more sources

Adaptor protein CIN85 potentiates the motility of osteosarcoma cells via the Akt/mTOR and MMP2‐COL3A1 axis

open access: yesMolecular Oncology, EarlyView.
CIN85 is highly expressed in osteosarcoma, particularly in metastatic lesions. Its overexpression increases cell migration and Matrigel invasion, while silencing CIN85 suppresses these behaviors. Transcriptome analysis shows that CIN85 regulates MMP2, COL3A1, and Akt/mTOR signaling. Targeting these pathways reverses CIN85‐induced motility, highlighting
Iryna Horak   +10 more
wiley   +1 more source

Los copistas y sus textos. Las traducciones moriscas del Corán en la Biblioteca Tomás Navarro Tomás (CSIC, Madrid)

open access: yesAl-Qantara : Revista de Estudios Arabes, 2014
Escondidas en una casa del pueblo de Almonacid de la Sierra (Aragón), de no más de 1600 habitantes, se nos han conservado treinta y siete copias del Corán y del tafsīr, copiadas por mudéjares y moriscos siendo once de ellas traducciones a la aljamía. Hoy
Nuria Martínez-de-Castilla-Muñoz
doaj   +1 more source

Hepatocellular carcinoma: Review of disease and tumor biomarkers. [PDF]

open access: yes, 2016
© The Author(s) 2016.Hepatocellular carcinoma (HCC) is a common malignancy and now the second commonest global cause of cancer death. HCC tumorigenesis is relatively silent and patients experience late symptomatic presentation. As the option for curative
Cox, IJ   +8 more
core   +1 more source

A novel quinazolinone insulin receptor inhibitor and its synergy with an EGFR inhibitor in glucose‐driven glioblastoma

open access: yesMolecular Oncology, EarlyView.
The novel styrylquinazolinone‐based molecule W1B effectively suppresses glioblastoma by inhibiting IGF1R and EGFR. In high‐glucose microenvironments driving tumor resistance, W1B acts synergistically with the EGFR inhibitor dacomitinib. This combination safely blocks compensatory survival signaling in zebrafish xenograft models. Showcasing promising in
Patryk Rurka   +9 more
wiley   +1 more source

Microprocessor Activity Controls Differential miRNA Biogenesis In Vivo

open access: yesCell Reports, 2014
In miRNA biogenesis, pri-miRNA transcripts are converted into pre-miRNA hairpins. The in vivo properties of this process remain enigmatic. Here, we determine in vivo transcriptome-wide pri-miRNA processing using next-generation sequencing of chromatin ...
Thomas Conrad   +3 more
doaj   +1 more source

Remembering Momigliano [PDF]

open access: yes, 2016
Obra ressenyada: T. CORNELL; O. MURRAY (eds.), The Legacy of Momigliano.
Sierra Martín, César
core  

Effects of Nanoparticle Geometry and Size Distribution on Diffusion Impedance of Battery Electrodes

open access: yes, 2012
The short diffusion lengths in insertion battery nanoparticles render the capacitive behavior of bounded diffusion, which is rarely observable with conventional larger particles, now accessible to impedance measurements. Coupled with improved geometrical
Bazant, M. Z., Song, J.
core   +1 more source

Transcriptomic-metabolomic reprogramming in EGFR-mutant NSCLC early adaptive drug escape linking TGFβ2-bioenergetics-mitochondrial priming. [PDF]

open access: yes, 2016
The impact of EGFR-mutant NSCLC precision therapy is limited by acquired resistance despite initial excellent response. Classic studies of EGFR-mutant clinical resistance to precision therapy were based on tumor rebiopsies late during clinical tumor ...
Bagai, Rakesh   +14 more
core   +1 more source

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