Correction to: "An Unusual Etiology of Recurrent Parathyroid Hormone-Independent Hypercalcemia". [PDF]
europepmc +1 more source
Putative α7-selective ligands interact with α9-containing nicotinic acetylcholine receptors and modulate immune functions of human mononuclear phagocytes. [PDF]
Mobasher M +10 more
europepmc +1 more source
The spectrum of Castleman diseases has expanded over the past three decades. The phenotype of the diseases varies not only among the three major types but also according to the patient ancestry. Summary Under the Castleman disease (CD) eponym, three distinct diseases sharing common pathological features have been described over time.
Eric Oksenhendler +4 more
wiley +1 more source
Utilizing Venturi effect for automated high-throughput droplet-MS from well plates.
Murray BE +5 more
europepmc +1 more source
Ginseng acetylcholine from various ginsengs and its <i>in vivo</i> physiological effect. [PDF]
Lee R, Kim JH, Kim JU, Hwang SH, Nah SY.
europepmc +1 more source
Preclinical models for evaluating psychedelics in the treatment of major depressive disorder
Psychedelic drugs have seen a resurgence in interest as a next generation of psychiatric medicines with potential as rapid‐acting antidepressants (RAADs). Despite promising early clinical trials, the mechanisms which underlie the effects of psychedelics are poorly understood.
Laith Alexander +5 more
wiley +1 more source
The role of the α7 nicotinic acetylcholine receptor in promoting M2 macrophage polarization at inflammatory sites. [PDF]
Mihara T +5 more
europepmc +1 more source
Non‐canonical PKG1 regulation in cardiovascular health and disease
It is well established that the cyclic GMP‐dependent protein kinase I (PKG1) is canonically activated by cyclic guanosine monophosphate (cGMP), enabling its regulation of vascular tone, cardiac function and smooth muscle homeostasis. However, diverse non‐canonical stimuli of PKG1 have also been identified.
Jie Su, Joseph Robert Burgoyne
wiley +1 more source
Presynaptic Terminal Proteins and Nicotinic Receptors Are Depleted from Mouse Parasympathetic Ganglionic Junctions Paralysed with Botulinum Neurotoxin Type A. [PDF]
Al-Sabi A, Lawrence GW.
europepmc +1 more source

