Results 21 to 30 of about 322,024 (266)

VEGFC/VEGFR3 Signaling‐Dependent Lymphatic Remodeling Modulates Cardiac Response to Pressure Overload

open access: yesJournal of the American Heart Association: Cardiovascular and Cerebrovascular Disease
Background Cardiac lymphangiogenesis has been proposed as a potential therapeutic target to prevent the development of heart failure. However, the efficiency of systemically delivered VEGFC (vascular endothelial growth factor C) protein in inducing ...
Lina Su   +13 more
doaj   +1 more source

Acute Myopericarditis Mimicking Acute Myocardial Infarction

open access: yesTürk Yoğun Bakim Derneği Dergisi, 2011
Acute coronary syndromes among young adults are relatively low when compared with older population in the intensive care unit. Electrocardiographic abnormalities mimicking acute coronary syndromes may be caused by non-coronary syndromes and the ...
Seval İzdeş   +4 more
doaj   +1 more source

Acute myocardial infarction hospital admissions and deaths in England: a national follow-back and follow-forward record-linkage study

open access: yesThe Lancet Public Health, 2017
Summary: Background: Little information is available on how primary and comorbid acute myocardial infarction contribute to the mortality burden of acute myocardial infarction, the share of these deaths that occur during or after a hospital admission ...
Perviz Asaria, MRCP   +6 more
doaj   +1 more source

Degradation mechanism of the von Willebrand factor A2 domain by nattokinase

open access: yesFEBS Letters, EarlyView.
Nattokinase, a natto‐derived protease, exhibits potent antithrombotic effects. This study demonstrates that nattokinase directly cleaves the von Willebrand factor (vWF) A2 domain in vitro. Unlike the native regulator ADAMTS13, nattokinase degrades folded vWF independently of shear stress.
Ryuichi Hyakumoto   +3 more
wiley   +1 more source

Elevated Levels of Anti-oxLDL Antibody in Relation with Acute Myocardial Infarction

open access: yesCentral Asian Journal of Medical Sciences, 2021
Objectives: We aimed to determine the serum level of anti-oxLDL antibody at the involving of acute myocardial infarction. Methods: In the case-control study, patients with acute myocardial infarction (coronary stenosis > 85%) as determined by quantities ...
Sumiya Tserendavaa   +2 more
doaj   +1 more source

Colchicine in Acute Myocardial Infarction [PDF]

open access: yesNew England Journal of Medicine
Inflammation is associated with adverse cardiovascular events. Data from recent trials suggest that colchicine reduces the risk of cardiovascular events.In this multicenter trial with a 2-by-2 factorial design, we randomly assigned patients who had myocardial infarction to receive either colchicine or placebo and either spironolactone or placebo.
Jolly, S.S.   +27 more
openaire   +4 more sources

A Complication of Acute Myocardial Infarction

open access: yesHCA Healthcare Journal of Medicine, 2020
Ventricular septal rupture (VSR) is a rare complication after acute myocardial infarction (MI). The case presents an apical four chamber echocardiogram showing mid-septal discontinuity with lateral to medial movement of the transected ventricular septum during systole and diastole.
Nakeya, Dewaswala, Robert Dean, Chait
openaire   +3 more sources

Cyclic azapeptide CD36 ligand attenuates cardiac injury and reduces long‐chain fatty acid accumulation after myocardial ischemia–reperfusion in mice

open access: yesFEBS Open Bio, EarlyView.
In a murine model of myocardial ischemia and reperfusion (MI/R), the CD36 azapeptide ligand MPE‐298 reduces cardiac injury and transiently lowers left ventricular long‐chain fatty acids (LCFAs) accumulation 3 h after reperfusion, accompanied by a decrease of oxidative stress and inflammation‐associated genes' expression in the heart and adipose tissue.
Jade Gauvin   +12 more
wiley   +1 more source

Acute myocardial infarction following a hornet sting [PDF]

open access: yesVojnosanitetski Pregled, 2009
Background. The occurrence of an acute myocardial infarction following a hornet sting has been very rarely reported in the previous literature. Pathogenetic mechanisms include direct action of the venom components on the coronary endothelium and allergic
Cvetković-Matić Danica   +6 more
doaj   +1 more source

From energy provision to protein synthesis: Tunnelling nanotubes as mediators of intercellular metabolic cooperation in cancer

open access: yesFEBS Open Bio, EarlyView.
The cytoskeleton‐mediated transport of mitochondria via tunnelling nanotubes restores respiration, increases ATP production, rescues cells from apoptosis, activates the AKT/mTOR signalling pathway, promotes cell migration and invasiveness, contributes to cancer progression and treatment resistance.
Stanislava Martínková, Jan Trnka
wiley   +1 more source

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