Effect of pro-inflammatory cytokines on urothelial cell adenosine triphosphate release and breakdown. [PDF]
Kaleska B +3 more
europepmc +1 more source
The pentaPod (P5) concept unites tridentate and tripodal fragments to form molybdenum and tungsten catalytic Chatt‐type complexes for N2‐to‐NH3 reduction. Two pentaPod ligands, P5Me and P5Pln, are available with either two dimethylphosphine or phospholano (Pln) groups. The complexes enable the investigation of reactivity differences in N2 reduction and,
Anna‐Marlene Vogt +3 more
wiley +1 more source
A clinical drug–drug interaction (DDI) study was designed to evaluate the effect of single and multiple oral doses of encorafenib on the single oral dose pharmacokinetics (PK) of the cytochrome P450 (CYP) enzyme probe substrates, losartan (CYP2C9), midazolam (CYP3A4), caffeine (CYP1A2), omeprazole (CYP2C19), and dextromethorphan (CYP2D6) administered ...
Joseph Piscitelli +6 more
wiley +1 more source
Bioinspired Fe<sub>3</sub>O<sub>4</sub>@Ag@ indocyanine green/adenosine triphosphate nanoenzyme in synergistic antibacterial performance. [PDF]
Hu D +8 more
europepmc +1 more source
Study of genetic polymorphisms of adenosine triphosphate-binding cassette B1 (ABCB1) gene in Iraqi nephrotic syndrome patients on prednisolone therapy [PDF]
Rawan Azad Mohammed +2 more
openalex +1 more source
Systemic aging fuels heart failure: Molecular mechanisms and therapeutic avenues
Abstract Systemic aging influences various physiological processes and contributes to structural and functional decline in cardiac tissue. These alterations include an increased incidence of left ventricular hypertrophy, a decline in left ventricular diastolic function, left atrial dilation, atrial fibrillation, myocardial fibrosis and cardiac ...
Zhuyubing Fang +7 more
wiley +1 more source
Clinical significance of splenic switch-off in adenosine triphosphate <sup>13</sup>N-ammonia positron emission tomography in patients without coronary artery disease. [PDF]
Ohara K +10 more
europepmc +1 more source
Abstract Aims Myocardial inflammation and impaired mitochondrial oxidative capacity are hallmarks of heart failure (HF) pathophysiology. The extent of myocardial inflammation in patients suffering from ischaemic cardiomyopathy (ICM) or dilated cardiomyopathy (DCM) and its association with mitochondrial energy metabolism are unknown.
Julius Borger +15 more
wiley +1 more source
Activation of Adenosine Triphosphate-Gated Purinergic 2 Receptor Channels by Transient Receptor Potential Vanilloid Subtype 4 in Cough Hypersensitivity. [PDF]
Li W +5 more
europepmc +1 more source
Abstract Background and aims The heart is a metabolic organ rich in mitochondria. The failing heart reprograms to utilize different energy substrates, which increase its oxygen consumption. These adaptive changes contribute to increased oxidative stress.
Qinghong Li +12 more
wiley +1 more source

