Bottom‐up Strategies for Generating Polymer Protocells That Mimic Cellular Communication
This review focuses on polymeric protocells produced using a bottom‐up approach. Polymer‐based assemblies guarantee stability and designability by adjusting the properties of the amphiphilic copolymers used. The review covers protocell architectures, production, and their intra‐ and intercellular communication mechanisms.
Gloria Saorin +3 more
wiley +1 more source
Effect of pro-inflammatory cytokines on urothelial cell adenosine triphosphate release and breakdown. [PDF]
Kaleska B +3 more
europepmc +1 more source
The pentaPod (P5) concept unites tridentate and tripodal fragments to form molybdenum and tungsten catalytic Chatt‐type complexes for N2‐to‐NH3 reduction. Two pentaPod ligands, P5Me and P5Pln, are available with either two dimethylphosphine or phospholano (Pln) groups. The complexes enable the investigation of reactivity differences in N2 reduction and,
Anna‐Marlene Vogt +3 more
wiley +1 more source
Comparative cardiovascular risk of sulfonylureas with low- and high-affinities for cardiac mitochondrial adenosine triphosphate-sensitive potassium channels versus dipeptidyl peptidase-4 inhibitors in patients with type 2 diabetes: A cohort study. [PDF]
Chen MH +8 more
europepmc +1 more source
Systemic aging fuels heart failure: Molecular mechanisms and therapeutic avenues
Abstract Systemic aging influences various physiological processes and contributes to structural and functional decline in cardiac tissue. These alterations include an increased incidence of left ventricular hypertrophy, a decline in left ventricular diastolic function, left atrial dilation, atrial fibrillation, myocardial fibrosis and cardiac ...
Zhuyubing Fang +7 more
wiley +1 more source
Potential effects of adenosine triphosphate and melatonin on oxidative and inflammatory optic nerve damage in rats caused by 5-fluorouracil. [PDF]
Somuncu AM +10 more
europepmc +1 more source
Abstract Aims Myocardial inflammation and impaired mitochondrial oxidative capacity are hallmarks of heart failure (HF) pathophysiology. The extent of myocardial inflammation in patients suffering from ischaemic cardiomyopathy (ICM) or dilated cardiomyopathy (DCM) and its association with mitochondrial energy metabolism are unknown.
Julius Borger +15 more
wiley +1 more source
Abstract Background and aims The heart is a metabolic organ rich in mitochondria. The failing heart reprograms to utilize different energy substrates, which increase its oxygen consumption. These adaptive changes contribute to increased oxidative stress.
Qinghong Li +12 more
wiley +1 more source
Bioinspired Fe<sub>3</sub>O<sub>4</sub>@Ag@ indocyanine green/adenosine triphosphate nanoenzyme in synergistic antibacterial performance. [PDF]
Hu D +8 more
europepmc +1 more source

