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Adhesins of Enterohemorrhagic Escherichia coli

EcoSal Plus, 2005
Enterohemorrhagic Escherichia coli (EHEC) was first recognized as a cause of human disease in 1983 and is associated with diarrhea and hemorrhagic colitis, which may be complicated by life-threatening renal and neurological sequelae.
Mark P, Stevens, Timothy S, Wallis
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Tissue interactions of Escherichia coli adhesins

Antonie van Leeuwenhoek, 1988
The E. coli adhesions show a remarkable tissue tropism in the human urinary tract. This obviously relates to the known compartmentation of glycoconjugates in the kidney. To function as a virulence factor in human urinary tract infections, an adhesin must evidently recognize such receptors at uroepithelia that are not excreted in soluble form in urine ...
T K, Korhonen   +8 more
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The Fimbrial Adhesins of Escherichia Coli

1987
Publisher Summary This chapter reviews the current knowledge of E. coli adhesions and their receptors. The main emphasis is given on the genetic and physiological aspects of adhesin production, the primary structure of adhesion subunits, their adhesive properties, and the characterization of adhesion receptors.
F K, De Graaf, F R, Mooi
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Adhesins of Escherichia coli

Acta Biotechnologica, 1990
AbstractE. coli has got increasing importance as a causative agent of intestinal and extra‐intestinal diseases. In both these infections adhesion of the bacteria to mucous surface cells are initial events for coionization and development of infection. Adhesins are bacterial recognition proteins which specifically interact with carbohydrate moieties of ...
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Fimbrial adhesins from extraintestinal Escherichia coli

Environmental Microbiology Reports, 2010
Summary Extraintestinal pathogenic Escherichia coli (ExPEC) represent an important subclass of E. coli that cause a wide spectrum of diseases in human and animal hosts. Fimbriae are key virulence factors of ExPEC strains.
Klemm, Per   +2 more
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Adhesins of Enteropathogenic Escherichia coli

EcoSal Plus, 2006
Enteropathogenic Escherichia coli (EPEC) strains induce morphological changes in infected epithelial cells. The resulting attaching and effacing (A/E) lesion is characterized by intimate bacterial adherence to epithelial cells, with microvillus destruction, cytoskeletal rearrangement, and aggregation of host ...
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