Results 111 to 120 of about 850 (150)
Incidence and risk factors of tocilizumab-induced hypofibrinogenemia in patients with thyroid eye disease: a single-center retrospective study. [PDF]
Wang E +6 more
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Congenital afibrinogenemia. A case report and therapeutic trials
S Ozsoylu, C Altay, B Corbacioğlu
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Late Intracerebral Hemorrhage After Successful Endovascular Closure of a Carotid-Cavernous Fistula: A Case Report and Updated Review. [PDF]
Uscamaita K +4 more
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Pediatrics, 1954
A case of congenital afibrinogenemia is described. Despite the absolute incoagulability of afibrinogenemic blood, this type of hemorrhagic diathesis is not accompanied by hemarthrosis. Systematic studies of this patient's blood revealed that all other known clotting factors are present in normal concentration.
P G, FRICK, I, McQUARRIE
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A case of congenital afibrinogenemia is described. Despite the absolute incoagulability of afibrinogenemic blood, this type of hemorrhagic diathesis is not accompanied by hemarthrosis. Systematic studies of this patient's blood revealed that all other known clotting factors are present in normal concentration.
P G, FRICK, I, McQUARRIE
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Acta Paediatrica, 1963
SUMMARYTwo patients, brother and sister, suffering from congenital afibrinogenemia are reported. This condition has previously not been described from Scandinavia. Both suffered from severe umbilical hemorrhage soon after birth and a continuing bleeding tendency.
S, OSEID, H M, SVENDSEN
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SUMMARYTwo patients, brother and sister, suffering from congenital afibrinogenemia are reported. This condition has previously not been described from Scandinavia. Both suffered from severe umbilical hemorrhage soon after birth and a continuing bleeding tendency.
S, OSEID, H M, SVENDSEN
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American Journal of Hematology, 1994
AbstractCongenital afibrinogenemia is a rare disorder with unusual clinical manifestations. The disease is inherited as an autosomal recessive trait and consanguinity is common among affected families. Clinical manifestations range from minimal bleeding to catastrophic hemorrhage.
H, al-Mondhiry, W C, Ehmann
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AbstractCongenital afibrinogenemia is a rare disorder with unusual clinical manifestations. The disease is inherited as an autosomal recessive trait and consanguinity is common among affected families. Clinical manifestations range from minimal bleeding to catastrophic hemorrhage.
H, al-Mondhiry, W C, Ehmann
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Thrombosis and Haemostasis, 1971
SummaryA case of congenital afibrinogenemia is reported.The patient is a 2 month old white female infant who presented bleeding from the umbilical stump a few days after birth. Subsequently other hemorrhagic manifestations were hematomas at the site of venipunctures.All routine plasmatic tests gave values greater than 1000 sec. The prothrombin time was
A Girolami, G Zacchello, R D’Elia
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SummaryA case of congenital afibrinogenemia is reported.The patient is a 2 month old white female infant who presented bleeding from the umbilical stump a few days after birth. Subsequently other hemorrhagic manifestations were hematomas at the site of venipunctures.All routine plasmatic tests gave values greater than 1000 sec. The prothrombin time was
A Girolami, G Zacchello, R D’Elia
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Congenital Afibrinogenemia in Goats
Zentralblatt für Veterinärmedizin Reihe A, 2010Summary A case of congenital afibrinogenemia in goats is described. The animal shows severe haemorrhagic diathesis with joint bleedings. The patient's blood was absolutely incoagulable. Immunologically no fibrinogen could be demonstrated in the plasma. The other clotting factors were present in normal concentrations.
H J, Breukink +4 more
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Acquired Afibrinogenemia in Pregnancy
New England Journal of Medicine, 1949ONE of the most catastrophic events in obstetric practice is the occurrence of a fulminating hemorrhagic diathesis usually associated with toxemia of pregnancy and toxic separation of the placenta. Although uncommon, this usually fatal disorder has been encountered by most obstetricians of long experience.1 2 3 For many years it has been suspected that
W C, MOLONEY, W J, EGAN, A J, GORMAN
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