Results 131 to 140 of about 1,453 (163)
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Aflatoxicosis in feedlot cattle
Journal of the American Veterinary Medical Association, 1985Aflatoxicosis was diagnosed in lightweight feedlot cattle fed aflatoxin-contaminated cottonseed or gin trash. Clinical signs of hepatic damage and death were recorded for more than 200 of the 14,000 animals in a feedlot. Aflatoxin concentration in feedlot products fed to these cattle ranged from 96 to 1,700 ng/g.
G D, Osweiler, D W, Trampel
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Acute aflatoxicosis: Case report
East African Medical Journal, 2005The objective of this presentation is to document the salient clinical findings in a case of aflatoxicosis and to review the literature on the same so as to increase the index of suspicion, enhance early diagnosis and improve management. The case was a 17-year-old schoolboy presenting with vomiting, features of infection and gastrointestinal tract ...
Mwanda O Walter. +2 more
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Aflatoxicosis in feeder cattle
Journal of the American Veterinary Medical Association, 1984Summary Aflatoxicosis was diagnosed as the cause of deaths in a group of crossbred feeder steers. Corn used in the ration being fed the steers was found to be contaminated with 1,500 ng of aflatoxin/g. Residues of the mycotoxin were detected in kidney tissue, and microscopic examination of liver sections revealed lesions typical of aflatoxicosis.
B M, Colvin +3 more
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Journal of the American Veterinary Medical Association, 1973
SUMMARY The aflatoxins are a group of toxic metabolites that vary in their toxic and carcinogenic potential The susceptibility of different animal species to aflatoxins also appears to be variable. In all animal species studied, hepatic damage is the principal injury induced.
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SUMMARY The aflatoxins are a group of toxic metabolites that vary in their toxic and carcinogenic potential The susceptibility of different animal species to aflatoxins also appears to be variable. In all animal species studied, hepatic damage is the principal injury induced.
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American Association of Bovine Practitioners Conference Proceedings, 1971
Aflatoxicosis is a noninfectious, noncontagious intoxication of an animal with a metabolite produced by a toxic mold. It is characterized in the acute phase by death loss and in the chronic cases by consumption decrease and death loss due to hepatic and renal insufficiency. Morbidity and mortality rates range from 5 to 55%. The etiological agents
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Aflatoxicosis is a noninfectious, noncontagious intoxication of an animal with a metabolite produced by a toxic mold. It is characterized in the acute phase by death loss and in the chronic cases by consumption decrease and death loss due to hepatic and renal insufficiency. Morbidity and mortality rates range from 5 to 55%. The etiological agents
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Companion Animal, 2021
Aflatoxins are toxic, naturally occurring bisfuranocoumarin compounds produced by certain strains of the moulds Aspergillus flavus, A. parasiticus and A. nomius. Aflatoxin metabolites cause hepatotoxicity by reacting with macromolecules (including DNA and proteins) to cause fatty liver or liver necrosis.
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Aflatoxins are toxic, naturally occurring bisfuranocoumarin compounds produced by certain strains of the moulds Aspergillus flavus, A. parasiticus and A. nomius. Aflatoxin metabolites cause hepatotoxicity by reacting with macromolecules (including DNA and proteins) to cause fatty liver or liver necrosis.
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Journal of the American Veterinary Medical Association, 1973
SUMMARY Aflatoxicosis caused by aflatoxin B1 and related toxins represents one of the most serious diseases of man, as well as of poultry, livestock, and other lower animals. The aflatoxins, produced by approximately 30% of the strains of Aspergillus flavus and by Penicillium puberulum, are frequent contaminants of harvested feeds and foods (rice, corn,
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SUMMARY Aflatoxicosis caused by aflatoxin B1 and related toxins represents one of the most serious diseases of man, as well as of poultry, livestock, and other lower animals. The aflatoxins, produced by approximately 30% of the strains of Aspergillus flavus and by Penicillium puberulum, are frequent contaminants of harvested feeds and foods (rice, corn,
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Altered lipid transport during aflatoxicosis
Toxicology and Applied Pharmacology, 1972The effects of graded doses of dietary aflatoxin (0, 0.625, 1.25, 2.5, 5.0 and 10.0 μg/g) on serum triglyceride fatty acids, serum total phospholipids and serum free and esterified cholesterol were measured in chickens. These classes of lipid were decreased significantly (P < 0.05) by a dose of 0.625 μg/g, and a dose of 2.5 μg/g was required to ...
H T, Tung, W E, Donaldson, P B, Hamilton
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Veterinary Research Communications, 1983
Under conditions presently prevailing in the UK aflatoxicosis in farm animals, as defined in this paper, is largely confined to cattle but acute and chronic clinical liver disease is seldom if ever encountered. There is circumstantial evidence for sub-clinical and non-specific aflatoxicosis but dose-effect relationships have yet to be established ...
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Under conditions presently prevailing in the UK aflatoxicosis in farm animals, as defined in this paper, is largely confined to cattle but acute and chronic clinical liver disease is seldom if ever encountered. There is circumstantial evidence for sub-clinical and non-specific aflatoxicosis but dose-effect relationships have yet to be established ...
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Concentrations of serum proteins during aflatoxicosis
Toxicology and Applied Pharmacology, 1975The concentrations of different classes of serum proteins of chickens to graded doses of dietary aflatoxin (0, 0.625, 1.25, 2.5, 5.0, and 10.0 μg/g of diet) were measured using disc gel electrophoresis. Total serum proteins were reduced significantly (p < 0.05) by a dose of 1.25 μg/g or greater. The α-globulins and β-globulins were reduced at levels of
H T, Tung +3 more
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