Results 251 to 260 of about 79,953 (279)
Case report: a case of AL amyloidosis with spontaneous giant retroperitoneal hematoma. [PDF]
Int J Emerg MedSheng Y, Zhu H.
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Safety and Efficacy of Teclistamab in Patients With Relapsed or Refractory AL Amyloidosis. [PDF]
Eur J HaematolStalker M +8 more
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Autologous stem cell transplantation in AL amyloidosis: Muddy waters. [PDF]
Blood RevHagen P, D'Souza A.
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New England Journal of Medicine
AL Amyloidosis A 58-year-old man presented with a 2-year history of eyelid lesions and several months of weight loss and fatigue. Scattered periorbital petechiae and purpura were noted, as well as coalescing, waxy papules on the eyelids.
Miguel Mansilla Polo +1 more
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AL Amyloidosis A 58-year-old man presented with a 2-year history of eyelid lesions and several months of weight loss and fatigue. Scattered periorbital petechiae and purpura were noted, as well as coalescing, waxy papules on the eyelids.
Miguel Mansilla Polo +1 more
+7 more sources
Once AL amyloidosis: not always AL amyloidosis
Amyloid, 2018Amyloid cardiomyopathy could be related to AL amyloidosis, wild-type transthyretin amyloidosis (ATTRwt) or hereditary amyloidosis (ATTRm).
Tulip Jhaveri +5 more
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Immunotherapy in AL Amyloidosis
Current Treatment Options in Oncology, 2022Light-chain amyloidosis is a rare disorder where a small clone of plasma cells is producing excess toxic light chains that deposit in various organs and cause dysfunction. Cardiac involvement is a major determinant of survival and rapid reduction of light chain is critical for recovery of organ function and overall survival. Immunotherapy targeting the
Yifei, Zhang, Raymond L, Comenzo
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The Lancet, 2014
A 62-year-old man came to our outpatient clinic with purpura on his eyelids. 7 months before, periocular purpura appeared after a minimal trauma. These lesions did not resolve for months. He also reported fatigue over the previous 6 months. On examination pinch purpura between smooth waxy papules were found (fi gure).
Barbara C, Böckle, Norbert T, Sepp
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A 62-year-old man came to our outpatient clinic with purpura on his eyelids. 7 months before, periocular purpura appeared after a minimal trauma. These lesions did not resolve for months. He also reported fatigue over the previous 6 months. On examination pinch purpura between smooth waxy papules were found (fi gure).
Barbara C, Böckle, Norbert T, Sepp
openaire +2 more sources
Primary generalized AL amyloidosis
Arkhiv patologii, 2021The paper describes an autopsy observation of a 71-year-old male with primary generalized AL amyloidosis lasting about 4 months after its manifestation to the onset of death from hepatic and renal failure. Total damage to the liver and spleen, as well as amyloid deposits in the kidneys, adrenal glands, and pancreas were noted.
E A, Kogan, B B, Saltykov, P V, Atanov
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La Revue de medecine interne, 2015
AL amyloidosis belongs to the group of conformational diseases. It is the most common type of amyloidosis with an estimated 500 new cases per year in France. It is due to a small and usually indolent plasma cell clone which synthesizes an unstable, misfolded monoclonal immunoglobulin light chain that is prone to aggregate and form amyloid fibrils.
A, Jaccard +3 more
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AL amyloidosis belongs to the group of conformational diseases. It is the most common type of amyloidosis with an estimated 500 new cases per year in France. It is due to a small and usually indolent plasma cell clone which synthesizes an unstable, misfolded monoclonal immunoglobulin light chain that is prone to aggregate and form amyloid fibrils.
A, Jaccard +3 more
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Blood, 2022
Abstract The treatment of patients with systemic light chain (AL) amyloidosis is a challenge to hematologists. Despite its generally small size, the underlying clone causes a rapidly progressing, often devastating multiorgan dysfunction through the toxic light chains that form amyloid deposits.
Giovanni Palladini, Giampaolo Merlini
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Abstract The treatment of patients with systemic light chain (AL) amyloidosis is a challenge to hematologists. Despite its generally small size, the underlying clone causes a rapidly progressing, often devastating multiorgan dysfunction through the toxic light chains that form amyloid deposits.
Giovanni Palladini, Giampaolo Merlini
openaire +3 more sources