Results 191 to 200 of about 33,271 (218)
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Characterization of the human aldolase B gene.

Molecular biology & medicine, 1986
The structure of the human gene encoding the aldolase B isozyme has been determined, including the sequence of 14,887 base-pairs. The 5'- and 3'-ends have been determined by S1 mapping. There is a single gene for this enzyme in humans that was determined from the sequence and restriction enzyme digestions of genomic DNA.
D R, Tolan, E E, Penhoet
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The ontogeny of fructose diphosphate aldolase B in the chick

Developmental Biology, 1972
Abstract The three homologous parental fructose-1, 6-diphosphate aldolases A, B, and C have previously been demonstrated in vertebrates. Although five-membered A–B, A–C, and B–C hybrid sets are readily produced in vitro by reversible dissociation of the appropriate parental enzymes, only A–B and A–C sets have been found heretofore in vertebrate ...
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The structural gene for aldolase B (ALDB) maps to 9q13→32

Annals of Human Genetics, 1985
SummaryWe used a cloned cDNA probe for the B subunit of human aldolase (ALDB) and Southern blotting techniques to analyse DNA from a series of rodent x human somatic cell hybrids for the presence of specific ALDB‐related sequences. Our results provide evidence for the assignment of the gene for ALDB to chromosome 9.
I, Henry   +8 more
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Human aldolase B gene: characterization of the genomic aldolase B gene and analysis of sequences required for multiple polyadenylations.

Journal of biochemistry, 1988
The chromosomal gene encoding human aldolase B was isolated. The gene is composed of nine exons interrupted by eight introns and spans 15 kb, and a single copy of it occurs per haploid human genome. The initiation of transcription occurs at three different sites.
T, Mukai   +5 more
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The aldolase inhibitor aldometanib mimics glucose starvation to activate lysosomal AMPK

Nature Metabolism, 2022
Chen-Song Zhang, Mengqi Li, Dong-Tai Liu
exaly  

Partial aldolase B gene deletions in hereditary fructose intolerance.

American journal of human genetics, 1990
Hereditary fructose intolerance (HFI) is an autosomal recessive condition caused by a deficiency of aldolase B. We have recently shown that three point mutations in this gene account for approximately 85% of HFI alleles in Europe and the United States and are thus of diagnostic importance.
N C, Cross, T M, Cox
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Aldolase B Deficiency

2009
Hubert Scharnagl   +199 more
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In vivo processing of rat aldolase B mRNA precursor.

Biochemistry international, 1987
Processed intermediates of aldolase B mRNA precursor in nuclei were analysed by hybridization with cDNA or intron-specific DNAs. The results indicated that there is, in part, a preferred order in the removal of eight introns from the primary transcript.
R, Tsutsumi, K, Tsutsumi, K, Ishikawa
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