Results 21 to 30 of about 33,271 (218)

The Expression of Aldolase B in Islets Is Negatively Associated With Insulin Secretion in Humans. [PDF]

J Clin Endocrinol Metab, 2018
Gerst F, Jaghutriz BA, Staiger H, Schulte AM, Lorza-Gil E, Kaiser G, Panse M, Haug S, Heni M, Schütz M, Stadion M, Schürmann A, Marzetta F, Ibberson M, Sipos B, Fend F, Fleming T, Nawroth PP, Königsrainer A, Nadalin S, Wagner S, Peter A, Fritsche A, Richter D, Solimena M, Häring HU, Ullrich S, Wagner R.   +27 more
europepmc   +2 more sources

Aldolase B inhibits metastasis through Ten-Eleven Translocation 1 and serves as a prognostic biomarker in hepatocellular carcinoma. [PDF]

Mol Cancer, 2015
Tao QF, Yuan SX, Yang F, Yang S, Yang Y, Yuan JH, Wang ZG, Xu QG, Lin KY, Cai J, Yu J, Huang WL, Teng XL, Zhou CC, Wang F, Sun SH, Zhou WP.   +16 more
europepmc   +3 more sources

Aldolase B suppresses hepatocellular carcinogenesis by inhibiting G6PD and pentose phosphate pathways

Nature Cancer, 2020
Shicheng Zhang, Yu Li, Jianping Ding
exaly   +2 more sources

Proteomics Used in Identifying Novel Correlates of Disease in Pediatric Metabolic Dysfunction‐Associated Steatotic Liver Disease [PDF]

Clinical and Translational Science
Metabolic dysfunction‐associated liver disease (MASLD) is a leading cause of liver disease in children. There is a paucity of data on potential biomarkers and therapeutic targets, especially in pediatric MASLD.
Diego Paine‐Cabrera, Lisa K. Harvey, Michele T. Pritchard, John Thyfault, Antonio Artigues, Udayan Apte, Voytek Slowik   +6 more
doaj   +2 more sources

Aldolase B Deficient Mice Are Characterized by Hepatic Nucleotide Sugar Abnormalities. [PDF]

J Inherit Metab Dis
Buziau AM, Lefeber DJ, Cassiman D, Rubio-Gozalbo ME, Kwast H, Tolan DR, Schalkwijk CG, Brouwers MCGJ.   +7 more
europepmc   +3 more sources

Hereditary Fructose Intolerance Diagnosed in Adulthood

Gut and Liver, 2021
Hereditary fructose intolerance (HFI) is an autosomal recessive disorder caused by a mutation in the aldolase B gene. HFI patients exhibit nausea, vomiting, abdominal pain, hypoglycemia, and elevated liver enzymes after dietary fructose exposure. Chronic
Min Soo Kim, Jin Soo Moon, Man Jin Kim, Moon-Woo Seong, Sung Sup Park, Jae Sung Ko   +5 more
doaj   +1 more source

Characterization of fructose-1,6-bisphosphate aldolase during anoxia in the tolerant turtle, Trachemys scripta elegans: an assessment of enzyme activity, expression and structure. [PDF]

PLoS ONE, 2013
One of the most adaptive facultative anaerobes among vertebrates is the freshwater turtle, Trachemys scripta elegans. Upon a decrease in oxygen supply and oxidative phosphorylation, these turtles are able to reduce their metabolic rate and recruit ...
Neal J Dawson, Kyle K Biggar, Kenneth B Storey   +2 more
doaj   +1 more source

Monoclonal antibodies against human aldolase B.

Kanzo, 1986
Masahiro ASAKA, Toshio KIMURA, Naoyuki HAYASHISHITA, Masao SAITO, Tamotsu MIYAZAKI, Shuichi KINO, Shinzo NISHI   +6 more
openaire   +3 more sources

Corrigendum: Unmet Psychosocial Needs of Patients Newly Diagnosed with Ulcerative Colitis: Results from the Nationwide Prospective Cohort Study in Korea

Gut and Liver, 2021
Hereditary fructose intolerance (HFI) is an autosomal recessive disorder caused by a mutation in the aldolase B gene. HFI patients exhibit nausea, vomiting, abdominal pain, hypoglycemia, and elevated liver enzymes after dietary fructose exposure. Chronic
Jung Rock Moon, Chang Kyun Lee, Sung Noh Hong, Jong Pil Im, Byong Duk Ye, Jae Myung Cha, Sung-Ae Jung, Kang-Moon Lee, Dong Il Park, Yoon Tae Jeen, Young Sook Park, Jae Hee Cheon, Hyesung Kim, BoJeong Seo, Youngdoe Kim, Hyo Jong Kim, the MOSAIK study group of the Korean Association for the Study of Intestinal Diseases (KASID)   +16 more
doaj   +1 more source

Aldolase B knockdown prevents high glucose-induced methylglyoxal overproduction and cellular dysfunction in endothelial cells. [PDF]

PLoS ONE, 2012
We used cultured endothelial cells as a model to examine whether up-regulation of aldolase B and enhanced methylglyoxal (MG) formation play an important role in high glucose-induced overproduction of advanced glycosylation endproducts (AGEs), oxidative ...
Jianghai Liu, Timothy Chun-Ping Mak, Ali Banigesh, Kaushik Desai, Rui Wang, Lingyun Wu   +5 more
doaj   +1 more source