Results 131 to 140 of about 66,158 (349)

Trigeminal‐mediated headshaking in horses

open access: yesEquine Veterinary Education, EarlyView.
Summary This clinical commentary is accompanying the manuscript on quantitative sensory testing and trigemino‐cervical thresholds in a Warmblood gelding affected by trigeminal‐mediated headshaking by Käfer‐Karrer et al. This new diagnostic method is promising for diagnosing a devastating disease which has an enormous impact on the horses welfare.
A. May, V. Franzen
wiley   +1 more source

Toll-like receptor signaling adapter proteins govern spread of neuropathic pain and recovery following nerve injury in male mice. [PDF]

open access: yes, 2013
BackgroundSpinal Toll-like receptors (TLRs) and signaling intermediaries have been implicated in persistent pain states. We examined the roles of two major TLR signaling pathways and selected TLRs in a mononeuropathic allodynia.MethodsL5 spinal nerve ...
A Ledeboer   +90 more
core   +1 more source

Differential Development of Facial and Hind Paw Allodynia in a Nitroglycerin-Induced Mouse Model of Chronic Migraine: Role of Capsaicin Sensitive Primary Afferents.

open access: yesBiological and Pharmaceutical Bulletin, 2018
Despite the relatively high prevalence of migraine or headache, the pathophysiological mechanisms triggering headache-associated peripheral hypersensitivities, are unknown.
Sol-Ji Kim   +6 more
semanticscholar   +1 more source

Insomnia symptoms amongst those with acute post‐traumatic headache attributed to mild traumatic brain injury

open access: yesHeadache: The Journal of Head and Face Pain, EarlyView.
Abstract Background Previous publications have described the presence and severity of insomnia amongst those with persistent post‐traumatic headache (PPTH), but there has been a paucity of studies investigating insomnia during the acute phase of post‐traumatic headache (APTH).
Ben J. Braunecker   +4 more
wiley   +1 more source

Contemporary views on inflammatory pain mechanisms: TRPing over innate and microglial pathways. [PDF]

open access: yes, 2016
Tissue injury, whether by trauma, surgical intervention, metabolic dysfunction, ischemia, or infection, evokes a complex cellular response (inflammation) that is associated with painful hyperalgesic states.
Guan, Zhonghui   +2 more
core   +2 more sources

Efficacy of Combined Versus Supplementary Injection Techniques With Inferior Alveolar Nerve Block for Mandibular Molars With Symptomatic Irreversible Pulpitis: A Systematic Review and Network Meta‐Analysis

open access: yesInternational Endodontic Journal, EarlyView.
ABSTRACT Background Achieving effective anaesthesia in mandibular molars with symptomatic irreversible pulpitis is challenging. Various strategies involving the inferior alveolar nerve block (IANB) have been investigated, such as supplementing with additional injections after IANB failure (SUPP) or combining injections with IANB as primary injections ...
Thanatpong Rujirawan   +2 more
wiley   +1 more source

Efficacy of Multi-Modal Migraine Prophylaxis Therapy on Hyperacusis Patients. [PDF]

open access: yes, 2020
ObjectivesTo evaluate the efficacy of a multi-modal migraine prophylaxis therapy for patients with hyperacusis.MethodsIn a prospective cohort, patients with hyperacusis were treated with a multi-modal step-wise migraine prophylactic regimen ...
Abouzari, Mehdi   +7 more
core  

Thalamo-cortical network activity between migraine attacks. Insights from MRI-based microstructural and functional resting-state network correlation analysis [PDF]

open access: yes, 2016
BACKGROUND: Resting state magnetic resonance imaging allows studying functionally interconnected brain networks. Here we were aimed to verify functional connectivity between brain networks at rest and its relationship with thalamic microstructure in ...
Colonnese, Claudio   +11 more
core   +1 more source

Role of the Excitability Brake Potassium Current IKD in Cold Allodynia Induced by Chronic Peripheral Nerve Injury

open access: yesJournal of Neuroscience, 2017
Cold allodynia is a common symptom of neuropathic and inflammatory pain following peripheral nerve injury. The mechanisms underlying this disabling sensory alteration are not entirely understood.
Alejandro González   +8 more
semanticscholar   +1 more source

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