Results 41 to 50 of about 42,096 (271)

Over diagnosis of bradykinin angioedema in patients treated with angiotensin-converting enzyme inhibitors or angiotensin II receptor blockers

open access: yesWorld Allergy Organization Journal, 2023
Background: Bradykinin angioedemas are a potentially serious side effect of angiotensin-converting enzyme inhibitors (ACEI) and more controversially of angiotensin II receptor blockers (ARB).
Marie Douillard, MD   +7 more
doaj   +1 more source

Classification, diagnosis, and approach to treatment for angioedema: Consensus report from the Hereditary Angioedema International Working Group [PDF]

open access: yes, 2014
Angioedema is defined as localized and self-limiting edema of the subcutaneous and submucosal tissue, due to a temporary increase in vascular permeability caused by the release of vasoactive mediator(s). When angioedema recurs without significant wheals,
Adam   +151 more
core   +1 more source

Subcutaneous C1‐Inhibitor Concentrate for prophylaxis during pregnancy and lactation in a patient with C1‐INH‐HAE

open access: yesClinical Case Reports, 2021
Subcutaneous plasma‐derived human C1‐Inhibitor concentrate (pdC1INH) may be safe and effective for long‐term prophylaxis during pregnancy and lactation in hereditary angioedema patients.
Shimalee Andarawewa   +1 more
doaj   +1 more source

Idiopathic Thrombocytopenic Purpura Misdiagnosed as Hereditary Angioedema [PDF]

open access: yes, 2015
Hereditary angioedema is a rare, but potentially life-threatening genetic disorder that results from an autosomal dominant trait. It is characterized by acute, recurrent attacks of severe local edema, most commonly affecting the skin and mucosa. Swelling
Andersen, Michelle Fog, Bygum, Anette
core   +5 more sources

Early diagnosis of hereditary angioedema in children: genetic testing should be prioritized

open access: yesAllergy, Asthma & Clinical Immunology
Background When a member of a family has been diagnosed with hereditary angioedema (HAE) before a child is born, the question of early diagnosis arises. Indeed, the first attacks may occur at birth.
A. Bocquet   +6 more
doaj   +1 more source

Terrible APS — a newly defined variant of severe APS

open access: yesFrontiers in Immunology
IntroductionAntiphospholipid syndrome (APS) presents with various clinical features and some patients exhibit progressive, refractory disease that does not meet the catastrophic APS (cAPS) criteria. This study describes a new subcategory of APS patients,
Stanley Niznik   +10 more
doaj   +1 more source

Disease Remission: A Scoping Review With Thematic Analysis of the Scientific Literature With Implications for Chronic Rhinosinusitis

open access: yesInternational Forum of Allergy &Rhinology, EarlyView.
ABSTRACT Background Remission, a term used to describe a goal of treatment for some chronic diseases, has recently been proposed for chronic rhinosinusitis (CRS). However, it is unclear what remission means for CRS and why it should serve as a goal in the present‐day treatment of CRS.
Nikhil Parail   +7 more
wiley   +1 more source

Simvastatin: A Risk Factor for Angioedema? [PDF]

open access: yes, 2013
Objective. To report a case of simvastatin-induced angioedema in a patient with near nightly episodes of orofacial angioedema. Case Summary. A 75-year-old African American female presented to the emergency department with recurrent face, lip, and tongue ...
Kara, Areeba   +2 more
core   +2 more sources

Interleukin‐5, Eosinophils, and Interleukin‐5 Pathway Inhibitors in Eosinophilic Granulomatosis With Polyangiitis

open access: yesArthritis &Rheumatology, EarlyView.
Interlukin‐5 (IL‐5) plays a crucial role in the pathogenesis of eosinophilic granulomatosis with polyangiitis (EGPA) by promoting eosinophil differentiation, activation, and survival. We present here a typical case of EGPA in which treatment with IL‐5 pathway inhibitors is prescribed, showing to be beneficial for the patient.
Alvise Berti, Christian Pagnoux
wiley   +1 more source

Management of hereditary angioedema with normal C1Inh: a series of 163 French patients

open access: yesOrphanet Journal of Rare Diseases
Background The diagnosis of hereditary angioedema with a normal C1Inh was genetic. The two most frequent pathogenic variants are found in the FXII and PLG genes.
Alexis Bocquet   +15 more
doaj   +1 more source

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