Results 251 to 260 of about 118,647 (313)
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Zeitschrift fur Kardiologie, 1993
Use of class-I antiarrhythmic agents (encainide, flecainide or moricizine) to suppress asymptomatic ventricular premature depolarizations does not decrease, but rather increases mortality from cardiac events after myocardial infarction. These patients should not be treated with antiarrhythmic drugs until improved survival is shown in a controlled ...
P, Honerjäger, G, Schmidt
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Use of class-I antiarrhythmic agents (encainide, flecainide or moricizine) to suppress asymptomatic ventricular premature depolarizations does not decrease, but rather increases mortality from cardiac events after myocardial infarction. These patients should not be treated with antiarrhythmic drugs until improved survival is shown in a controlled ...
P, Honerjäger, G, Schmidt
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[Combined action of anti-arrhythmia agents].
Biulleten' eksperimental'noi biologii i meditsiny, 1991A significant potentiation of antiarrhythmic effect was observed in 121 dogs with arrhythmias 24 and 48 hours after the coronary artery ligation when the following drugs were combined: N-propylajmaline bromide (1A class) and trimecaine (1B class), quinidine (1A class) and trimecaine, N-propylajmaline bromide and anaprilin (2 class). The potentiation is
E I, Gendenshteĭn +3 more
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[Arrhythmogenic effects of anti-arrhythmia agents].
Zeitschrift fur die gesamte innere Medizin und ihre Grenzgebiete, 1987Arrhythmogenic effects appear in 5-22% of all patients. On the basis of a literary investigation on 11,547 patients with 1,114 proarrhythmic effects and of 21 own observations mechanisms, risks and diagnostics of this dangerous side effect are demonstrated.
D, Pfeiffer +4 more
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[The cardiotoxic properties of anti-arrhythmia agents].
Eksperimental'naia i klinicheskaia farmakologiia, 1997Experiments were conducted on cats to study the cardiotoxic and arrhythmogenic properties of some antiarrhythmic agents in continuous intravenous infusion in doses multiple of the dose which causes an antiarrhythmic effect on an acontic model of arrhythmia in rats in 50% of cases.
A A, Spasov, N A, Gurova
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[Propafenone--a promising anti-arrhythmia agent?].
Sbornik vedeckych praci Lekarske fakulty Karlovy univerzity v Hradci Kralove. Supplementum, 1991Propafenone hydrochloride (Rytmonorm, Prolekofen) belongs to the membranous anti-arrhythmical agents of IC group. Simultaneously, it produces a mild beta blocking effect and blocks slow Calcium channels. The mentioned agent is of current use in treating both the supraventricular and ventricular tachyarrhythmias.
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[Exogenous adenosine as an anti-arrhythmia agent].
Zeitschrift fur Kardiologie, 1997Adenosine has potent cardiac electrophysiologic effects including a negative chronotropic action on the sinus node and a predominant negative dromotropic action on the AV node. The latter property has mainly led to the use of adenosine as antiarrhythmic agent for the acute management of paroxysmal supraventricular tachycardia (PSVT) mediated by a ...
J, Tebbenjohanns +5 more
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[Current classification of anti-arrhythmia agents].
Zeitschrift fur Kardiologie, 2000Antiarrhythmic drugs can be divided into four Vaughan Williams classes (I-IV) according to defined electrophysiological effects on the myocardium. Thus, the Vaughan Williams classification also coincides with the main myocardial targets of the antiarrhythmics, i.e., myocardial sodium-, potassium-, and calcium-channels or beta-adrenergic receptors.
J, Weirich, W, Wenzel
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[Cardiotropic effect of anti-arrhythmia agents].
Farmakologiia i toksikologiia, 1979In experiments on contractile proteins isolated from the myocardium rather than from the musculus gastrocnemius it was shown that in contradistinction to lidocain the drugs quinine, quinidine, novocainamide, papaverin, obsidan and isoptin at a concentration of 3 .
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