Results 131 to 140 of about 8,392 (217)

Serum Levels of Coagulation Factors in Patients with Inflammatory Bowel Disease. [PDF]

open access: yesIran J Pathol
Memar B   +4 more
europepmc   +1 more source
Some of the next articles are maybe not open access.

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Familial Functional Antithrombin III Deficiency

Scandinavian Journal of Haematology, 1980
A family with a tendency to thrombosis and decreased antithrombin III (AT III) activity in plasma, but normal immunoreactive AT III is reported. 7 members of the family had the AT III defect, 4 of whom have had thrombotic episodes. The importance of biological determination of AT III when studying patients with recurrent thrombotic episodes is ...
J Dyerberg   +2 more
exaly   +3 more sources

Hereditary antithrombin III deficiency. Effect of antithrombin III deficiency on platelet function

The American Journal of Medicine, 1976
Antithrombin III (AT III) is the main physiologic inhibitor of thrombin, and activated factors X and IX as well. Normal levels of AT III appear to be necessary to maintain blood fluidity and to prevent thrombosis. Four families with AT III deficiency and recurrent venous thromboembolism have been reported on. We present an additional family with AT III
A, Carvalho, L, Ellman
semanticscholar   +4 more sources

Antithrombin III concentrate: its catabolism in health and in antithrombin III deficiency

Scandinavian Journal of Clinical and Laboratory Investigation, 1981
The catabolism of purified radiolabelled antithrombin III (AT III) concentrate was studied in two normals and two patients with congenital AT III deficiency both alone and combined with warfarin. The radiolabelling with iodine monochloride did not change the quality of the concentrate. The half-life varied between 3.4 and 4.8 days.
L Tengborn, I M Nilsson
exaly   +3 more sources

Familial Antithrombin III Deficiency

open access: yesQJM: An International Journal of Medicine, 1982
SUMMARY Antithrombin III is the major physiological inhibitor of the coagulation mechanism and a deficiency of this protein results in a marked predisposition to venous thromboembolic disease. Three Scottish families with a deficiency of this protein are described and other reported families are reviewed.
J. Winter   +6 more
openaire   +2 more sources

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