Results 151 to 160 of about 8,392 (217)

Neonatal antithrombin III deficiency

The American Journal of Medicine, 1989
Hemorrhagic and thrombotic complications are common in sick preterm infants and may reflect inadequate regulation of coagulation. All neonates have low levels of the pivotal regulator antithrombin III (ATIII) compared with adults. Plasma levels of ATIII are very low in preterm infants and are further diminished in infants with respiratory distress ...
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Pathophysiology of Antithrombin III Deficiency

Veterinary Clinics of North America: Small Animal Practice, 1988
This article focuses on the pathophysiology of thrombosis in patients with acquired antithrombin III deficiency. Antithrombin III is an important natural inhibitor of the hemostatic mechanism, and a hypercoagulable state is often induced in diseases causing antithrombin III deficiency.
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Venous Thrombosis: Antithrombin III Deficiency

Critical Care Nursing Clinics of North America, 1993
Antithrombin (ATIII) is believed to be one of the body's most powerful natural anticoagulants, so individuals with a deficiency of this protein exhibit a marked propensity to venous thrombosis. This article will focus on the pathophysiology of this disorder, including clinical presentation, diagnosis, and treatment.
M, Halfman, D E, Berg
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Congenital antithrombin III deficient neonate treated with antithrombin III concentrates

Thrombosis Research, 1993
A patient with antithrombin III deficiency developed deep vein thrombosis during her first pregnancy. Her pregnancy and delivery were successfully treated with simultaneous administration of antithrombin III concentrates and low molecular weight heparin. She delivered a 2,412g girl at 39 weeks' gestation. The baby was administered with antithrombin III
A, Shiozaki, T, Arai, R, Izumi, K, Niiya, N, Sakuragawa   +4 more
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Antithrombin III Deficiency in a Japanese Family

Thrombosis and Haemostasis, 1979
Antithrombin III has been reported to be decreased in the cases of several thrombotic disorders and the decreased Antithrombin III is known to induce the hypercoagulable state. This study was started from the 28 year-old male patient who developed the superior sagital sinus thrombosis after appendectomy and it was followed by deep vein thrombosis of ...
M. Nakagawa, T. Kawamura, H. Tsuji, Y. Okajima, S. Urano, T. Kitani, H. Iiichi   +6 more
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Familial antithrombin III deficiency.

Lancet (London, England), 1983
Antithrombin III is the major physiological inhibitor of the coagulation mechanism and a deficiency of this protein results in a marked predisposition to venous thromboembolic disease. Three Scottish families with a deficiency of this protein are described and other reported families are reviewed.
J H, Winter, A, Fenech, W, Ridley, B, Bennett, A M, Cumming, M, Mackie, A S, Douglas   +6 more
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Antithrombin III deficiency in ischaemic stroke

Klinische Wochenschrift, 1983
AT III activity and concentration were measured in 36 patients (mean age 65.5 yrs, range 43-77 yrs) with ischaemic stroke within maximally 48 h of the acute event. In 12 patients (= 33%) AT III activity was reduced below 18.4 IU/ml: 50% of these patients showed normal and 50% reduced AT III concentration of less than 22 mg/dl.
V, Hossmann, W D, Heiss, H, Bewermeyer
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ANTITHROMBIN III DEFICIENCY IN A CHINESE FAMILY

Medical Journal of Australia, 1981
Familial antithrombin III deficiency, which is inherited by autosomal dominant transmission, is now well recognized as a cause of recurrent venous thromboembolism. Many such families have now been described, but few came from Asia. This report details a Chinese kindred with three members living in Sydney, two of whom are affected.
A J, Concannon, J, Low
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Inherited Antithrombin III Deficiency in the Neonate

Archives of Pediatrics & Adolescent Medicine, 1994
To describe two cases of inherited antithrombin III (AT-III) deficiency presenting at less than or equal to 28 days of age, and to review other neonatal reports.Clinical descriptions of two patients and literature review of known references to the neonatal presentation of this disorder.Academic neonatal intensive care unit.Case reports--two patients ...
J, Seguin, K, Weatherstone, C, Nankervis
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Inherited deficiency of antithrombin III in an Italian kindred

La Ricerca in Clinica e in Laboratorio, 1980
Life-long symptoms of venous thromboembolism were present throughout five generations in a large Italian kindred. Eight out of the 22 tested members of the family had reduced plasma levels of antithrombin III (AT III), the main physiological inhibitor of blood coagulation. Three of them, aged 2-28 years, but no thrombotic manifestations.
A. Tripodi, C. Boyer, M. T. Canciani, A. D'Angelo, P. M. Mannucci   +4 more
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