Results 141 to 150 of about 8,392 (217)
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Antithrombin III deficiency in an indian family
Thrombosis Research, 1982D Mohanty, Kanjaksha Ghosh, G Garewal
exaly +3 more sources
Molecular Heterogeneity of Inherited Antithrombin III Deficiency
New England Journal of Medicine, 1983Edward V Prochownik +2 more
exaly +2 more sources
Hereditary antithrombin iii deficiency and thromboembolic disease
American Journal of Hematology, 1976J J Veltkamp
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Thrombosis Research, 1987
Antithrombin III (AT III) microheterogeneity was investigated in 12 cases of congenital AT III deficiency and 2 cases of congenital AT III abnormality by isoelectric focusing (IEF) and immunofixation. In congenital AT III deficiency, IEF and immunofixation revealed AT III as 8 bands which was indistinguishable from normal control in terms of the number
N, Sakuragawa +4 more
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Antithrombin III (AT III) microheterogeneity was investigated in 12 cases of congenital AT III deficiency and 2 cases of congenital AT III abnormality by isoelectric focusing (IEF) and immunofixation. In congenital AT III deficiency, IEF and immunofixation revealed AT III as 8 bands which was indistinguishable from normal control in terms of the number
N, Sakuragawa +4 more
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Congenital antithrombin III deficiency
The American Journal of Medicine, 1989Jack Hirsh, Franco Piovella, Mario Pini
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Myocardial infarction in a neonate with hereditary antithrombin III deficiency
Acta Paediatrica, International Journal of Paediatrics, 1993S Peeters +2 more
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Aortic Thrombosis in Antithrombin III Deficiency
JAMA: The Journal of the American Medical Association, 1982To the Editor.— Although we are told in the article by Michael E. Shapiro, MD, and Edwin W. Salzman, MD (1981;245:1759), that abnormal antithrombin III (AT III) levels were documented in both cases weeks to months after any short-term event or surgery, in case 1 the reduced AT III level was found at a time when resting pain had been present for three ...
J H, Winter, B, Bennett, A S, Douglas
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Blood Reviews, 1988
A moderate reduction of plasma antithrombin activity is an uncommon but clinically important cause of severe thromboembolic disease. In recent years the molecule responsible for the major part of this activity (antithrombin III) has been extensively characterised and the mode of inheritance of familial deficiencies worked out.
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A moderate reduction of plasma antithrombin activity is an uncommon but clinically important cause of severe thromboembolic disease. In recent years the molecule responsible for the major part of this activity (antithrombin III) has been extensively characterised and the mode of inheritance of familial deficiencies worked out.
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Heterogeneity of the "Classical" Antithrombin III Deficiency
Thrombosis and Haemostasis, 1980SummaryWe investigated two thrombophilia families with the "classical" type of antithrombin III deficiency, i.e., with a low antithrombin III level measured both by immunochemical and functional methods.We obtained different antithrombin III patterns in the plasma of the affected members of the two families with the modified two dimensional ...
G, Sas +4 more
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Journal of the American Veterinary Medical Association, 1982
The hypercoagulable state accompanying nephrotic syndrome in 3 dogs was characterized by markedly increased fibrinogen and factor VIII concentrations. Antithrombin III was mildly reduced in 1 dog (74% of normal) and markedly reduced in the other 2 dogs (
Green Ra, Kabel Al
semanticscholar +1 more source
The hypercoagulable state accompanying nephrotic syndrome in 3 dogs was characterized by markedly increased fibrinogen and factor VIII concentrations. Antithrombin III was mildly reduced in 1 dog (74% of normal) and markedly reduced in the other 2 dogs (
Green Ra, Kabel Al
semanticscholar +1 more source