Results 151 to 160 of about 1,433 (211)
Dabigatran Attenuates the Binding of Thrombin to Platelets-A Novel Mechanism of Action. [PDF]
Lindahl TL +7 more
europepmc +1 more source
Recommendations on Monitoring and Replacement of Antithrombin, Fibrinogen, and Von Willebrand Factor in Pediatric Patients on Extracorporeal Membrane Oxygenation: The Pediatric Extracorporeal Membrane Oxygenation Anticoagulation CollaborativE Consensus Conference. [PDF]
Zantek ND +13 more
europepmc +1 more source
Dynamics of coagulation proteins upon ICU admission and after one year of recovery from COVID-19: a preliminary study. [PDF]
Behar-Lagares R +11 more
europepmc +1 more source
Purification of Two Progressive Antithrombins of Human Plasma
U Abildgaard
exaly +2 more sources
Antithrombin was purified from Bothrops jararaca plasma by affinity chromatography using HiTrap Heparin HP column, and its molecular weight, amino-terminal sequence, carbohydrate content, isoelectric point, inhibition of bovine thrombin, and ...
Anita Mitico Tanaka-Azevedo
exaly +2 more sources
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Thrombosis Research, 1987
Antithrombin III (AT III) microheterogeneity was investigated in 12 cases of congenital AT III deficiency and 2 cases of congenital AT III abnormality by isoelectric focusing (IEF) and immunofixation. In congenital AT III deficiency, IEF and immunofixation revealed AT III as 8 bands which was indistinguishable from normal control in terms of the number
N, Sakuragawa +4 more
openaire +2 more sources
Antithrombin III (AT III) microheterogeneity was investigated in 12 cases of congenital AT III deficiency and 2 cases of congenital AT III abnormality by isoelectric focusing (IEF) and immunofixation. In congenital AT III deficiency, IEF and immunofixation revealed AT III as 8 bands which was indistinguishable from normal control in terms of the number
N, Sakuragawa +4 more
openaire +2 more sources
QUANTITATIVE EFFECTS OF IMMEDIATE ANTITHROMBINS
Anthony J. Glazko, John H. Ferguson
exaly +2 more sources
Molecular Medicine Today, 1995
Antithrombin, the main inhibitor of thrombosis in blood, is bound and activated by the heparin-like side-chains that line the small vasculature. We now have good depictions of the heparin-binding site on antithrombin, and of the way in which mutations at this site cause thrombotic disease.
R, Carrell +3 more
openaire +2 more sources
Antithrombin, the main inhibitor of thrombosis in blood, is bound and activated by the heparin-like side-chains that line the small vasculature. We now have good depictions of the heparin-binding site on antithrombin, and of the way in which mutations at this site cause thrombotic disease.
R, Carrell +3 more
openaire +2 more sources
Thrombosis Research, 2018
Antithrombin is a key endogenous anticoagulant whose deficiency constitutes a strong risk factor for thrombosis. The study of antithrombin deficiency has generated excellent, and in some cases, surprising results that may be extrapolated to other thrombophilia and genetic disorders.
Javier, Corral +2 more
openaire +2 more sources
Antithrombin is a key endogenous anticoagulant whose deficiency constitutes a strong risk factor for thrombosis. The study of antithrombin deficiency has generated excellent, and in some cases, surprising results that may be extrapolated to other thrombophilia and genetic disorders.
Javier, Corral +2 more
openaire +2 more sources

