Results 191 to 200 of about 37,019 (222)
Research progress of aquaporin 1 and 4 in diabetic retinopathy
Xiaojuan Zhang +3 more
openalex +2 more sources
Aquaporin 1 Is Overexpressed in Lung Cancer and Stimulates NIH-3T3 Cell Proliferation and Anchorage-Independent Growth [PDF]
Mohammad Obaidul Hoque +12 more
openalex +1 more source
Viticulture is facing increasing challenges due to climate change. The focus on fast growth and sweet berries has come at the expense of stress resilience. Grafting onto Phylloxera‐resistant rootstocks from American species has been the most successful form of ecological pest management.
Samia Daldoul +6 more
wiley +1 more source
Functional Analysis of Aquaporin-1 Deficient Red Cells [PDF]
John Mathai +8 more
openalex +1 more source
Pilocarpine activates M1 and M3 muscarinic receptors in porcine ciliary muscle, engaging calcium‐dependent phospholipase C (PLC), nitric oxide synthase (NOS), and cyclooxygenase (COX) pathways that increase prostaglandin E2 (PGE2) synthesis. Beyond nitric oxide (NO) modulation, PGE2 emerges as a key mediator linking muscarinic signaling to ciliary ...
Giovanna Benozzi +3 more
wiley +1 more source
Expression and roles of aquaporin 1 in hippocampus of mice model with traumatic brain injury
Bo Qiu +4 more
openalex +1 more source
Vasopressin-induced serine 269 phosphorylation reduces Sipa1l1 (signal-induced proliferation-associated 1 like 1)-mediated aquaporin-2 endocytosis [PDF]
Po-Jen Wang +6 more
openalex +1 more source
Protective Effect of Pediococcus pentosaceus Li05 on Constipation via TGR5/TPH1/5‐HT Activation
The mechanism of Li05 in alleviating constipation. Li05 intervention regulated gut microbiota and bile acid metabolism, increasing 5‐HT levels through the TGR5/TPH1 activation to ameliorate constipation. ABSTRACT Pediococcus pentosaceus Li05, a strain of lactic acid bacteria isolated from the faeces of healthy volunteers, exhibited potential protective
Hui Chen +8 more
wiley +1 more source
NFAT5 Regulates IL8 to Promote Cell Growth and Migration in Non‐Small Cell Lung Cancer
Proposed mechanism for the role of NFAT5 in NSCLC progression. NFAT5 could promote cell growth and motility by regulating IL8 directly in NSCLC cell lines. Elevated IL8 expression caused enhancing NF‑κB signaling pathway partially through autocrine or paracrine effects. ABSTRACT Background In certain types of solid tumors, nuclear factor of activated T
Jinliang Chen +4 more
wiley +1 more source

