Results 221 to 230 of about 158,510 (246)

Steroids in Early ARDS

Chest, 2007
2007;132;1094 Chest Jan G. Zijlstra and Jack J. Ligtenberg Steroids in Early ARDS http://chestjournal.chestpubs.org/content/132/3/1094.full.html and services can be found online on the World Wide Web at: The online version of this article, along with updated information ISSN:0012-3692 ) http://chestjournal.chestpubs.org/site/misc/reprints.xhtml ( of ...
Jan G. Zijlstra, Jack J. M. Ligtenberg
openaire   +4 more sources

Corticosteroids in ARDS

Chest, 2007
2007;132;1093-1094; Chest Carolyn S. Calfee and Michael A. Matthay G. Umberto Meduri, Paul E. Marik, Stephen M. Pastores, Djillali Annane, Corticosteroids in ARDS: A Counterpoint http://chestjournal.org and services can be found online on the World Wide Web at: The online version of this article, along with updated information ). ISSN: 0012-3692. http:/
Paul E. Marik, Djillali Annane, G. Umberto Meduri, Stephen M. Pastores   +3 more
openaire   +3 more sources

Mechanism of ARDS

2000
Initially described more than 25 years ago [1] in 12 patients, adult respiratory distress syndrome (ARDS) is classically defined as an acute, severe alteration in lung structure and function characterized by hypoxemia, low-compliance lungs with low functional residual capacity, and diffuse radiographic infiltrates due to increased lung microvascular ...
BRAZZI, Luca, Pelosi P, Gattinoni L.
openaire   +3 more sources

Neuromuscular Blockers and ARDS

New England Journal of Medicine, 2010
patients with the acute respiratory distress syndrome (ARDS) treat-ed early with cisatracurium had improved 90-day survival and an increased number of ventilator-free days. We are concerned about a number of methodologic limitations that detract from the reliability of the findings. First, considering that patients who are not paralyzed can trigger the
Zudin Puthucheary, Hugh Montgomery, Nicholas Hart   +2 more
openaire   +4 more sources

ROS Signaling in the Pathogenesis of Acute Lung Injury (ALI) and Acute Respiratory Distress Syndrome (ARDS)

Advances in Experimental Medicine and Biology, 2017
The generation of reactive oxygen species (ROS) plays an important role for the maintenance of cellular processes and functions in the body. However, the excessive generation of oxygen radicals under pathological conditions such as acute lung injury (ALI)
M. Kellner, S. Noonepalle, Qing Lu, A. Srivastava, E. Zemskov, S. Black   +5 more
semanticscholar   +1 more source

Akutes Lungenversagen (ARDS)

1997
Beim ARDS (fruher: „adult respiratory distress syndrome“) handelt es sich um eine akute, schwere pulmonale Insuffizienz als typische Reaktion der Lunge auf unterschiedliche Noxen. Das ARDS ist keine Krankheitseinheit, sondern ein entzundliches Syndrom der Lunge, gekennzeichnet durch eine diffuse alveolare Schadigung und eine gesteigerte Permeabilitat ...
Reinhard Larsen, Thomas Ziegenfuß
openaire   +2 more sources

Intensivmedizinisches Management des ARDS / Management of ARDS

2001
Das schwere, akute Lungenversagen (ARDS) ist durch eine meist generalisierte Inflammation der Lunge charakterisiert, die zu einer ausgepragten Gasaustauschstorung, einem Permeabilitatsodem und einer Anderung der Lungendehnbarkeit fuhrt. Wichtigste aktuelle Prinzipien der intensivmedizinischen Behandlung des ARDS sind eine lun-genprotektive Beatmung mit
M. Schutz, K. J. Falke, U. Kaisers
openaire   +2 more sources

Echocardiography in ARDS

2020
RV failure occurred in around 22% of moderate to severe ARDS, even when ventilated with a lung protective approach. Risk factors are a plateau pressure ≥27 cmH2O, a driving pressure ≥18 cm H2O, a PaCO2 ≥48 mmHg, a PaO2/FiO2
openaire   +2 more sources

When ARDS May Not Be ARDS

A48. CRITICAL CARE CASE REPORTS: CAUSES AND COMPLICATIONS OF ACUTE RESPIRATORY FAILURE, 2019
K. Ducey, T.J.K. Vossler, S.A. Nonas
openaire   +1 more source

Hemodynamics and Therapy in Ards

1989
ARDS patients suffer from diffuse injury of the pulmonary microvasculature with increased permeability to plasma proteins. A definitive diagnosis of ARDS requires evidence of the following: a) increased venous admixture \( ({\rm{e}}{\rm{.g}}{\rm{.,\dot QS/\dot QT > 30}}\% {\rm{ or P}}_a {\rm{O}}_2 /{\rm{F}}_{{\rm{I }}} {\rm{O}}_2 < 250); \)
openaire   +3 more sources