Results 31 to 40 of about 299,530 (310)

Repurposing Interleukin-6 Inhibitors to Combat COVID-19. [PDF]

open access: yes, 2020
Severe acute respiratory syndrome-coronavirus-2 (SARS-CoV-2) is a pandemic with major implications across the world. One of the most frequent causes of death from SARS-CoV-2 is fatal pneumonia from coronavirus disease 2019 (COVID-19), which is associated
Kato, Shumei, Kurzrock, Razelle
core   +1 more source

Acute Pancreatitis and COVID-19: A Single-Center Experience

open access: yesJournal of Multidisciplinary Healthcare, 2021
Anna Annunziata,1 Antonietta Coppola,1 Paolo Andreozzi,2 Maurizia Lanza,1 Francesca Simioli,1 Novella Carannante,3 Camilla Di Somma,1 Pierpaolo Di Micco,4 Giuseppe Fiorentino1 1Department of Critic Area, I Unit COVID, Monaldi-Cotugno Hospital, Naples ...
Annunziata A   +8 more
doaj  

Host–Viral Interactions Revealed among Shared Transcriptomics Signatures of ARDS and Thrombosis: A Clue into COVID-19 Pathogenesis

open access: yesTH Open, 2020
Severe novel corona virus disease 2019 (COVID-19) infection is associated with a considerable activation of coagulation pathways, endothelial damage, and subsequent thrombotic microvascular injuries.
Aastha Mishra   +2 more
doaj   +1 more source

High Flow Nasal Cannula Oxygen Therapy in Long Hauler Covid-19 Patients

open access: yesSouth East Asia Nursing Research, 2021
Long Hauler Covid-19 is a condition that describes a person who has recovered from Covid-19 and is declared with a negative PCR smear 2 times but still feels signs and symptoms for a longer period of time, even severe and critical symptoms.
Sarinti Sarinti, Fefi Eka Wahyuningsih
doaj   +1 more source

In vitro and in vivo effects of salbutamol on neutrophil function in acute lung injury [PDF]

open access: yes, 2006
Background: Intravenous salbutamol (albuterol) reduces lung water in patients with the acute respiratory distress syndrome (ARDS). Experimental data show that it also reduces pulmonary neutrophil accumulation or activation and inflammation in ARDS ...
McAuley, Daniel F.   +4 more
core   +2 more sources

Oxygenation inhibits the physiological tissue-protecting mechanism and thereby exacerbates acute inflammatory lung injury [PDF]

open access: yes, 2005
Acute respiratory distress syndrome (ARDS) usually requires symptomatic supportive therapy by intubation and mechanical ventilation with the supplemental use of high oxygen concentrations.
  +62 more
core   +5 more sources

Rationale for Prolonged Glucocorticoid Use in Pediatric ARDS: What the Adults Can Teach Us. [PDF]

open access: yes, 2016
Based on molecular mechanisms and physiologic data, a strong association has been established between dysregulated systemic inflammation and progression of acute respiratory distress syndrome (ARDS).
Meduri, Gianfranco Umberto   +1 more
core   +1 more source

Sarcoidosis Presenting as Acute Respiratory Distress Syndrome. [PDF]

open access: yes, 2018
Sarcoidosis is a multisystem granulomatous disease of unknown origin. It typically involves the lungs and mediastinal lymph nodes in a chronic fashion. However, acute syndrome has been reported possibly in response to systemic release of proinflammatory ...
Chan, Vincent   +3 more
core   +2 more sources

Respiratory Mechanics and Gas Exchange in Acute Respiratory Distress Syndrome Associated with COVID-19

open access: yesОбщая реаниматология, 2022
Aim. To compare respiratory mechanics and gas exchange in patients with acute respiratory distress syndrome (ARDS) with and without COVID-19.Material and methods. We examined 96 patients, who were divided into two groups.
R. A. Ibadov   +4 more
doaj   +1 more source

Epigenetic contribution of the myosin light chain kinase gene to the risk for acute respiratory distress syndrome [PDF]

open access: yes, 2017
Acute respiratory distress syndrome (ARDS) is a devastating clinical syndrome with a considerable case fatality rate (∼30%-40%). Health disparities exist with African descent (AD) subjects exhibiting greater mortality than European descent (ED ...
Fortman, Jeffrey D.   +6 more
core   +1 more source

Home - About - Disclaimer - Privacy