Arginase-2 enhances monocyte-endothelial interaction through regulation of integrin and membrane receptor expression: role in atherogenesis. [PDF]
Ajalbert G +9 more
europepmc +1 more source
Pre-emptive treatment in later-onset urea cycle disorders: a clinical perspective on glycerol phenylbutyrate. [PDF]
Spada M, Porta F.
europepmc +1 more source
Amino Acid Metabolism in Health and Disease. [PDF]
Su Z +7 more
europepmc +1 more source
The competition of amino acid among tumors, tumor-associated macrophages and T cells based on metabolic reprogramming. [PDF]
Xianyu C +6 more
europepmc +1 more source
Notch-1 induces M2 polarization of tumor-associated macrophages via the YAP/EZH2/FOXD1/CD47 signaling axis, thereby driving non-small cell lung cancer progression. [PDF]
Guo L, Liu W, Shan M, Liu Y.
europepmc +1 more source
Arginase I mRNA Therapy – A Novel Approach to Rescue Arginase 1 Enzyme Deficiency [PDF]
Arginase I (ARG1) deficiency is an autosomal recessive urea cycle disorder, caused by deficiency of the enzyme Arginase I, resulting in accumulation of arginine in blood. Current Standard of Care (SOC) for ARG1 deficiency in patients or those having detrimental mutations of ARG1 gene is diet control.
Romesh Subramanian, Christopher Cheng
exaly +3 more sources
Related searches:
Case Series of Arginase 1 Deficiency: Expanding the Spectrum in Hyperargininemia
Pediatrics International, 2022AbstractBackgroundArginase‐1 deficiency is a rare, autosomal recessively inherited disorder of the urea cycle. In this study, we describe the clinical and molecular details of six patients who were diagnosed with argininemia, and we describe two of the patients with hyperargininemia who carried two novel variations of the Arginase‐1 gene.MethodsThe ...
Cengiz Havalı, Sevil Dorum
exaly +3 more sources

