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Management of reactive arthritis
Expert Opinion on Pharmacotherapy, 2004Reactive arthritis (ReA) is an aseptic form of articular inflammation induced by infections mainly localised in the gastrointestinal (enteroarthritis) or urogenital (uroarthritis) tracts. The bacteria principally involved as causative agents are Chlamydia, Salmonella, Shigella, Campylobacter and Yersinia.
Carlo, Palazzi +4 more
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Best Practice & Research Clinical Rheumatology, 2011
Reactive arthritis (ReA) can be defined as the development of sterile inflammatory arthritis as a sequel to remote infection, often in the gastrointestinal or urogenital tract. Although no generally agreed-upon diagnostic criteria exist, the diagnosis is mainly clinical, and based on acute oligoarticular arthritis of larger joints developing within 2-4
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Reactive arthritis (ReA) can be defined as the development of sterile inflammatory arthritis as a sequel to remote infection, often in the gastrointestinal or urogenital tract. Although no generally agreed-upon diagnostic criteria exist, the diagnosis is mainly clinical, and based on acute oligoarticular arthritis of larger joints developing within 2-4
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Reactive arthritis and psittacosis
The American Journal of Medicine, 1986A 54-year old man had severe inflammatory polyarthritis 10 days after the onset of an acute febrile illness that was serologically documented to be psittacosis. The pattern and chronicity of the articular symptoms, the response to nonsteroidal anti-inflammatory agents, and the presence of HLA-B7-CREG strongly suggest that this was a reactive arthritis.
S M, Cooper, J A, Ferriss
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Poststreptococcal reactive arthritis
Current Opinion in Rheumatology, 2000The occurrence of an entity designated poststreptococcal reactive arthritis (PSReA) has been highlighted in recent reports. The syndrome was considered part of the spectrum of acute rheumatic fever by some, whereas others stressed the differences between the two diseases.
E M, Ayoub, H A, Majeed
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Infectious Disease Clinics of North America, 2017
Reactive arthritis is classified as a spondyloarthropathy. Current concepts of disease suggest an infectious trigger, followed by inflammatory arthritis. Several mechanisms have been proposed to explain the interaction of host susceptibility and microorganism.
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Reactive arthritis is classified as a spondyloarthropathy. Current concepts of disease suggest an infectious trigger, followed by inflammatory arthritis. Several mechanisms have been proposed to explain the interaction of host susceptibility and microorganism.
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Poststreptococcal reactive arthritis
Current Opinion in Rheumatology, 2002Poststreptococcal reactive arthritis (PSRA) refers to a poststreptococcal arthritic condition that does not fulfill the Jones Criteria for diagnosis of acute rheumatic fever. Clinical features include additive rather than migratory arthritis that responds relatively poorly to salicylates and nonsteroidals; persistence for mean of 2 months; elevated ...
Stanford T, Shulman, Elia M, Ayoub
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Pathogenesis of Reactive Arthritis
Current Rheumatology Reports, 2001There is good evidence that bacteria persist in vivo in patients with reactive arthritis (ReA). While Chlamydia seem to hide inside the joint, other areas such as gut mucosa or lymph nodes seem to be more likely places for Salmonella and Yersinia. T-helper (Th) 1 cells secreting cytokines such as IFN gamma and TNF alpha are crucial for an effective ...
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Poststreptococcal Reactive Arthritis
Journal of the American Podiatric Medical Association, 2006Acute rheumatic fever is a delayed inflammatory disease that follows streptococcal infection of the throat. Poststreptococcal reactive arthritis is a sterile arthritis associated with antecedent streptococcal infection in patients not fulfilling the Jones criteria for acute rheumatic fever. Poststreptococcal reactive arthritis has been reported to have
Daniel, Logan, Patrick J, McKee
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Rheumatology, 1983
A review of Yersinia reactive arthritis is presented with a description of clinical manifestations and immunological features. The pathogenesis of the reactive lesion has so far not been resolved since no conclusive data on the existence or nonexistence of Yersinia antigens in the inflamed joints have been reported.
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A review of Yersinia reactive arthritis is presented with a description of clinical manifestations and immunological features. The pathogenesis of the reactive lesion has so far not been resolved since no conclusive data on the existence or nonexistence of Yersinia antigens in the inflamed joints have been reported.
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Orthopaedic Nursing, 2004
What do conjunctivitis, urethritis/cervicitis, and arthritis have in common? These are all clues to the diagnosis of reactive arthritis. It takes a keen eye, a thorough history, a good physical examination, and a broad differential diagnosis to pull together the picture when sorting through seemingly unrelated clinical symptoms.
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What do conjunctivitis, urethritis/cervicitis, and arthritis have in common? These are all clues to the diagnosis of reactive arthritis. It takes a keen eye, a thorough history, a good physical examination, and a broad differential diagnosis to pull together the picture when sorting through seemingly unrelated clinical symptoms.
openaire +2 more sources