Results 41 to 50 of about 160,290 (294)

Causes of attention deficit hyperactivity disorder [PDF]

open access: yes, 2000
Plan BAs a society we often look at how we can fix current problems. This study takes another approach that has not been as well researched in the area of Attention Deficit Hyperactivity Disorder, and that is the potential causes of this highly diagnosed
Mertig, Daniel A.
core  

Prevalence and contributing factors to attention deficit hyperactivity disorder:a study of five- to fifteen-year-old children in Zhabei District, Shanghai [PDF]

open access: yes, 2014
INTRODUCTION: This work aims to understand the features among 5- to 15-year-old children with attention deficit hyperactivity disorder (ADHD) in Zhabei District in Shanghai. METHODS: Children with ADHD were studied using general background questionnaire,
Coghill, David   +7 more
core   +1 more source

Mechanoluminescent HOF Nanotransducers Enabled Sono‐Optogenetics in Parkinsonian Rats

open access: yesAdvanced Functional Materials, EarlyView.
We present a mechanoluminescent system utilizing porous hydrogen‐bonded organic frameworks (HOFs) as a toolkit for focused ultrasound‐triggered, non‐invasive light delivery to the deep brain in rats. This approach enables the specific activation of PV‐GPe neurons in dopamine‐depleted Parkinson's disease rat models, resulting in a comparable alleviation
Wenliang Wang   +18 more
wiley   +1 more source

Attention-Deficit/Hyperactivity Disorder [PDF]

open access: yesArchives of Neurology, 2002
Attention-deficit/hyperactivity disorder (ADHD) is one of the most common conditions demanding the time and knowledge of school nurses. Although the condition was identified 100 years ago, it is only in the past decade that information has been clarified. New medication is continually being developed, and the area of comorbidity is exploding.
  +7 more sources

An examination of the effects of stimulant medication on response inhibition: A comparison between children with and without attention deficit hyperactivity disorder

open access: yes, 2011
This study investigated whether methylphenidate is effective in improving response inhibition in children with Attention Deficit Hyperactivity Disorder (ADHD).
Rachel Brackenridge   +7 more
core   +1 more source

Plugging the Attention Deficit: Perceptual Load Counters Increased Distraction in ADHD. [PDF]

open access: yes, 2013
Objective: Increased vulnerability to extraneous distraction is a key symptom of Attention-Deficit Hyperactivity Disorder (ADHD), which may have particularly disruptive consequences.
Forster, S   +14 more
core   +1 more source

Nigra‐Subthalamic Dopaminergic Circuitry Modulates and Represents Distinct Pain Modality in Physiological and Pain States in Mice

open access: yesAdvanced Science, EarlyView.
Nigral dopaminergic (DA) neurons modulate and represent pain with a preference to a particular modality (mechanical) and laterality (contralateral), which are controlled by nigral GABAergic neurons. The pain modulation is mimicked by the nigro‐subthalamic projection and its downstream neurons, involving D2‐like receptors.
Ying Ji   +13 more
wiley   +1 more source

Executive functioning in adults with attention deficit hyperactivity disorder: A systematic review

open access: yesActa Neurológica Colombiana
Introduction: Executive function deficits are associated with attention deficit hyperactivity disorder (ADHD) in both children and adults. The inattentive subtype is primary related to “cold” executive functions, while the hyperactive subtype is related
Carlos Francisco Rincón   +2 more
doaj   +1 more source

An Activity‐Dependent NEPAS–PTX3 Axis Links Neurovascular and Myelin Deficits to Cognitive Impairment

open access: yesAdvanced Science, EarlyView.
An activity‐dependent pathway links prefrontal circuit hypoactivity to cognitive impairment. Reduced PVA–mPFC activity upregulates NEPAS, which suppresses PTX3 secretion, leading to impaired angiogenesis, myelin deficits, and memory decline. Rescue is achieved by NEPAS knockdown or chemogenetic circuit activation.
Boya Hu   +11 more
wiley   +1 more source

mGluR5 in ECCCK to BLA Circuit Modulates Depressive‐Like Phenotypes through CCK Signaling

open access: yesAdvanced Science, EarlyView.
Dysregulation of mGluR5 and CCK signaling contributes to major depressive disorder, yet circuit‐level mechanisms remain unclear. Here, the ECCCK→BLA pathway is identified as a critical regulator of affective behavior. mGluR5 modulates synaptic function and CCK signaling within this circuit, controlling stress susceptibility and depressive‐like states ...
Muhammad Asim   +4 more
wiley   +1 more source

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