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Autoimmune Encephalitis

Pediatrics In Review, 2022
Autoimmune encephalitis is a common and treatable cause of encephalitis in children and adults. Individuals present with a variety of symptoms, including altered mental status, behavioral changes, irritability, insomnia, developmental regression, seizures, dyskinetic movements, and autonomic instability.
Saba, Jafarpour, Jonathan D, Santoro
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Autoimmune Encephalitis

Zhurnal nevrologii i psikhiatrii im. S.S. Korsakova, 2015
This chapter gives an overview of the syndromes associated with antibodies directed to membrane-bound or synaptic proteins. In contrast to the classical syndromes, disease occurs in younger patients as well; patients are also seen by non-neurologists like psychiatrists and pediatricians, and patients tend to have a more favorable response to ...
Van Sonderen, Agnes   +1 more
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Management of autoimmune encephalitis

Current Opinion in Neurology, 2021
Purpose of review Autoimmune encephalitides are established diagnoses in contemporary neurology. Their management poses a regular challenge for almost all neurologists. One may ask if the concept of 1st line and 2nd line treatment is still up to date, which new data on the antibody-defined encephalitis types exist, and how to ...
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Immunotherapy in autoimmune encephalitis

Current Opinion in Neurology, 2022
Purpose of review Autoimmune encephalitis (AE) refers to immune-mediated neurological syndromes often characterised by the detection of pathogenic autoantibodies in serum and/or cerebrospinal fluid which target extracellular epitopes of neuroglial antigens.
Benjamin P, Trewin   +3 more
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Neurobiology of autoimmune encephalitis

Current Opinion in Neurobiology, 2018
Autoimmune encephalitis presenting with amnesia, seizures, and psychosis is highly topical in basic and clinical neuroscience. Recent studies have identified numerous associated autoantibodies, targeting cell-surface synaptic proteins including neurotransmitter receptors (e.g. NMDA receptors (NMDARs)) and a secreted protein, LGI1.
Masaki Fukata   +3 more
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