Results 171 to 180 of about 207,009 (370)
(4) AUTOIMMUNITY AND RENAL DISEASES
Seiichi Shibata, Toshihiko Nagasawa
openalex +2 more sources
The therapeutic potential of ApoVs derived from stem cells from human exfoliated deciduous teeth (SHED‐ApoVs) in the treatment of CD8+ T cell‐mediated hypersensitivity reactions is noteworthy. SHED‐ApoVs are capable of fusing with the plasma membrane of CD8+ T cells, which subsequently triggers a series of events characterized by calcium overload ...
Anqi Liu+13 more
wiley +1 more source
Tolerance and autoimmunity to erythroid differentiation (B-G) major histocompatibility complex alloantigens of the chicken. [PDF]
C Havele+2 more
openalex +1 more source
Functional Polarization of Liver Macrophages by Glyco Gold Nanoparticles
Macrophages play a critical role in several pathophysiological diseases by being involved in the activation of adaptive and innate immune responses. The repolarization of macrophages using gold nanoparticles functionalized with glycans for a more selective targeting toward a restorative phenotype leads to an amelioration of the events.
Jennifer Fernandez Alarcon+25 more
wiley +1 more source
Human FOXP3+ Regulatory T Cell Heterogeneity and Function in Autoimmunity and Cancer.
J. Wing, A. Tanaka, S. Sakaguchi
semanticscholar +1 more source
Artesunate Inhibits Neointimal Hyperplasia by Promoting IRF4 Associated Macrophage Polarization
This study shows that macrophage interferon regulatory factor 4 (IRF4) improved arterial injury‐induced neointimal hyperplasia by promoting of M2 polarization via up‐regulating krüppel‐like factor 4 (KLF4) in rodent and nonhuman primate models. Notably, artesunate is identified as a potent inducer of IRF4 in macrophages, and proposed as a promising ...
Jinlin Miao+15 more
wiley +1 more source
DIFFERING IMMUNE MECHANISMS TRIGGERING AUTOIMMUNITY IN PRIMARY SCLEROSING CEOLANGITIS (PSC) AND AUTOIMMUNE CHRONIC ACTIVE HEPATITIS (aCAH) OF CHILDHOOD [PDF]
G Miell-Vergani+3 more
openalex +1 more source
LACK156‐173 is internalized into RA‐FLSs via CAPN2‐mediated endocytosis and inhibits the aggressive phenotype of RA‐FLSs by restoring dysregulated fatty acid synthesis metabolism mediated by overexpressed FASN. The findings suggest that targeting the restoration of fatty acid metabolism could potentially alleviate synovial invasion and joint damage in ...
Jianling Su+17 more
wiley +1 more source