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Abortive infection of Japanese quail cells with avian sarcoma viruses.
Virology, 1972Abstract The replication of Prague strains of Rous sarcoma virus (PR RSV) and avian sarcoma virus B77 in Japanese quail cells is abortive in a quantitative sense. Although efficient infection of quail cells, as evidenced by transformation, was obtained with these viruses, the yield of progeny was suppressed by 102-fold or more relative to the chick ...
R. Friis
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Enhancement and inhibition of avian sarcoma viruses by polycations and polyanions.
Virology, 1969Abstract Polycations enhance the infectivity of avian sarcoma viruses for chick embryo fibroblast cultures up to 80-fold. The enhancement is restricted to members of avian tumor virus subgroups B, C, and D and to RSV(O). Subgroup A viruses are either unaffected or inhibited by polycations.
K. Toyoshima, Peter K. Vogt
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Temperature-sensitive avian sarcoma viruses: a physiological comparison of twenty mutants.
Virology, 1973Abstract The behavior of 20 temperature-sensitive mutants of avian sarcoma virus has been examined. Three general parameters of infection were studied: virus replication, ability of the virus to induce cell focus formation on a monolayer and ability of the virus to induce cell colony formation in agar suspension.
J. A. Wyke, M. Linial
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Virology, 1976
Five coordinate temperature-sensitive mutants of avian sarcoma viruses which fail to transform or produce infectious progeny at 41° have been analyzed by genetic recombination. Four, namely LA334, 336, 338, and 343, carry multiple mutations. One of these mutations is always in the src gene affecting initiation and maintenance of transformation.
D. Blair, W. Mason, E. Hunter, P. Vogt
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Five coordinate temperature-sensitive mutants of avian sarcoma viruses which fail to transform or produce infectious progeny at 41° have been analyzed by genetic recombination. Four, namely LA334, 336, 338, and 343, carry multiple mutations. One of these mutations is always in the src gene affecting initiation and maintenance of transformation.
D. Blair, W. Mason, E. Hunter, P. Vogt
semanticscholar +3 more sources
[32] Biological techniques for avian sarcoma viruses
, 1979Publisher Summary This chapter describes a quantitative and reproducible assay of Rous sarcoma virus (RSV), the focus assay, and methods for obtaining cloned preparations of RSV, through both the focus and soft agar-colony assays. Large-scale growth of RSV is discussed in the chapter.
E. Hunter
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Conditional lethal mutants of avian sarcoma viruses. I. Physiology of ts 75 and ts 149.
Virology, 1971R. Friis, K. Toyoshima, P. Vogt
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Characteristics of cellular RNA related to the transforming gene of avian sarcoma viruses.
Cell, 1978Deborah H. Spector +3 more
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Virus Research, 1988
Twelve independent isolates of avian sarcoma viruses (ASVs) can be divided into four groups according to the transforming genes harbored in the viral genomes. The first group is represented by viruses containing the transforming sequence, src, inserted in the viral genome as an independent gene; the other three groups of viruses contain transforming ...
L H, Wang, H, Hanafusa
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Twelve independent isolates of avian sarcoma viruses (ASVs) can be divided into four groups according to the transforming genes harbored in the viral genomes. The first group is represented by viruses containing the transforming sequence, src, inserted in the viral genome as an independent gene; the other three groups of viruses contain transforming ...
L H, Wang, H, Hanafusa
openaire +2 more sources