Results 181 to 190 of about 7,370 (219)
Cobalt Toxicity Induces Retinopathy and Optic Neuropathy in Mice. [PDF]
Invest Ophthalmol Vis SciObied B +8 more
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Betaine alleviates deficits in motor behavior, neurotoxic effects, and neuroinflammatory responses in a rat model of demyelination. [PDF]
Toxicol RepEssawy AE +4 more
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Journal of Neurobiology, 1973
AbstractThe transport of RNA from the ganglion cell bodies within the retina to the contralateral optic tectum has been studied in the chick following intraocular injection of radioactive uridine. By tracing the appearance of labeled RNA at the proximal end of the optic nerve as it leaves the eyeball and comparing this to the time of arrival of RNA ...
S C, Bondy, C J, Madsen
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AbstractThe transport of RNA from the ganglion cell bodies within the retina to the contralateral optic tectum has been studied in the chick following intraocular injection of radioactive uridine. By tracing the appearance of labeled RNA at the proximal end of the optic nerve as it leaves the eyeball and comparing this to the time of arrival of RNA ...
S C, Bondy, C J, Madsen
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Simulation of axoplasmic transport
Journal of Theoretical Biology, 1984We have analysed a kinetic model of axonal transport by simulating experimental tracer profiles. The existence of three phases of axoplasmic transport is assumed: fast anterograde, slow anterograde and retrograde. Each phase has its characteristic velocity. Transported materials are postulated to shift between these phases.
T, Takenaka, H, Gotoh
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Axoplasmic Transport and Neurological Surgery
Neurosurgery, 1982Both the slow bulk flow (1 mm/day) of cytoplasm and the rapid transport of specific small molecules, as well as macromolecules (at rates of up to 410 mm/day), are known to occur in the neuronal cell body, axon, and dendrites. The basic phenomena as well as mechanisms of these transport phenomena and their implication for a variety of pathophysiological
B H, Smith, P L, Kornblith
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Axoplasmic transport in aged rats
Experimental Neurology, 1982Abstract The velocities of 3H-proteins undergoing fast axoplasmic transport in sciatic nerves were measured in rats of different ages with close attention paid to temperature regulation during the period of transport. The rate of axoplasmic transport slowed from a high of 453 ± 15 mm/day at 3 months of age, to 406 ± 16 mm/day in the 37- to 38-month ...
D P, Stromska, S, Ochs
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1975
Publisher Summary The existence of axoplasmic flow—that is, the longitudinal translocation of materials in axons, is generally recognized. This chapter describes axoplasmic flow in the nerves of mature animals. Such nerves are usually opaque and axoplasmic flow in them eludes direct observations. Indirect methods have to be used to detect the flow in
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Publisher Summary The existence of axoplasmic flow—that is, the longitudinal translocation of materials in axons, is generally recognized. This chapter describes axoplasmic flow in the nerves of mature animals. Such nerves are usually opaque and axoplasmic flow in them eludes direct observations. Indirect methods have to be used to detect the flow in
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Optic nerve axoplasm and papilledema
Survey of Ophthalmology, 1975A detailed review of optic nerve axoplasm is presented. A number of hypotheses have been postulated for the pathogenesis of papilledema associated with increased intracranial pressure. These hypotheses, mechanical and nonmechanical, are critically evaluated in relation to five essential features of papilledema.
J D, Wirtschafter +2 more
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Axoplasmic transport of muscarinic receptors
Nature, 1980The reality of axoplasmic transport is widely accepted; various neutrotransmitters, enzymes, labelled proteins and peptides are known to move rapidly along the axons of different nerve fibres. In the terminals of sympathetic nerves, noradrenaline release is controlled by various regulatory mechanisms which imply the occurrence of presynaptic receptors.
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RAPID AXOPLASMIC TRANSPORT IN DYSTROPHIC MICE
Journal of Neurochemistry, 1977Abstract— Rapid axoplasmic transport was studied in dystrophic mice of the 129/ReJ‐dy strain. Proteins transported in vivo through α‐motoneurons of the sciatic nerve were labeled by injections of [3H] or [35S] amino acids into the ventral horn of the lumbar spinal cord.
Mclane, J A, Mcclure, W O
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