Homozygous deletions localize novel tumor suppressor genes in B-cell lymphomas [PDF]
Integrative genomic and gene-expression analyses have identified amplified oncogenes in B-cell non-Hodgkin lymphoma (B-NHL), but the capability of such technologies to localize tumor suppressor genes within homozygous deletions remains unexplored.
Agirre-Ena, X. (Xabier) +20 more
core
306 | CENTRAL NERVOUS SYSTEM RELAPSE IN HIGH‐GRADE B‐CELL LYMPHOMA WITH MYC AND BCL2 AND/OR BCL6 REARRANGEMENTS: A MULTICENTER STUDY FROM THE SPANISH GROUP GELTAMO [PDF]
Cristina García Herce +37 more
openalex +1 more source
B-cell non-Hodgkin's lymphoma cell line (Karpas 1106) with complex translocation involving 18q21.3 but lacking BCL2 rearrangement and expression [PDF]
E Nacheva +9 more
openalex +1 more source
Super-enhancer hypermutation alters oncogene expression in B cell lymphoma
E. Bal +15 more
semanticscholar +1 more source
Osteosarcoma is the most common primary bone tumor with limited treatment options and a terrible prognosis. This review provides a comprehensive summary of the recent development of osteoimmunomodulatory implants for post‐operative osteosarcoma treatment, of which the potential utility in evoking durable anti‐osteosarcoma immunity and accelerating bone
Yilong Dong +6 more
wiley +1 more source
Prevalence profile of cutaneous T- and B-cell lymphoma in Gharbia Governorate, Egypt
Background Lymphomas represent a heterogeneous group of lymphoid malignancies with varied patterns of clinical behavior and responses to treatment. The WHO classification distinguishes lymphoid neoplasms derived from precursor lymphoid cells from those ...
Nagwa M. Elwan +3 more
doaj +1 more source
Circulating CD44 and intercellular adhesion molecule‐1 levels in low grade non‐Hodgkin lymphoma and B‐cell chronic lymphocytic leukemia patients during interferon‐α‐2a treatment [PDF]
Meral Beksaç +5 more
openalex +1 more source
Genetics and Pathogenesis of Diffuse Large B‐Cell Lymphoma
R. Schmitz +39 more
semanticscholar +1 more source
Streptococcus anginosus extracellular vesicles (SA‐EVs) accumulate in gastric tissue, disrupt epithelial tight junctions, and induce gastritis characterized by neutrophil infiltration and elevated cytokines (TNF‐α, IL‐6, IL‐17A). Proteomics identifies TMPC and FBP62 as key SA‐EVs virulence factors; their genetic deletion attenuates inflammation ...
Ying Gong +12 more
wiley +1 more source

