Single amino acid-based PROTACs trigger degradation of the oncogenic kinase BCR-ABL in chronic myeloid leukemia (CML). [PDF]
Zhang J+5 more
europepmc +1 more source
Transformation of Myeloid Leukemia Cells to Cytokine Independence by Bcr-Abl Is Suppressed by Kinase-defective Hck [PDF]
Jack M. Lionberger+2 more
openalex +1 more source
[Corrigendum] ND‑09 inhibits chronic myeloid leukemia K562 cell growth by regulating BCR‑ABL signaling. [PDF]
Liu YH, Zhu M, Lei PP, Pan XY, Ma WN.
europepmc +1 more source
The HSP90 inhibitor KW-2478 depletes the malignancy of BCR/ABL and overcomes the imatinib-resistance caused by BCR/ABL amplification. [PDF]
Zeng D+7 more
europepmc +1 more source
T cell receptors specific for an imatinib-induced mutation in BCR-ABL for adoptive T cell therapy. [PDF]
Hsu MT+3 more
europepmc +1 more source
[Comparison of BCR::ABL (P210) mRNA levels detected by dPCR and qPCR methods in patients with chronic myeloid leukemia]. [PDF]
Gao HL+6 more
europepmc +1 more source
CDK8/19 inhibition attenuates G1 arrest induced by BCR-ABL antagonists and accelerates death of chronic myelogenous leukemia cells. [PDF]
Khamidullina AI+17 more
europepmc +1 more source
BCR-ABL triggers a glucose-dependent survival program during leukemogenesis through the suppression of TXNIP. [PDF]
Feng L+14 more
europepmc +1 more source