Results 171 to 180 of about 60,584 (224)

Correction: Weber et al. Stat5 Exerts Distinct, Vital Functions in the Cytoplasm and Nucleus of Bcr-Abl⁺ K562 and Jak2(V617F)⁺ HEL Leukemia Cells. <i>Cancers</i> 2015, <i>7</i>, 503-537. [PDF]

Cancers (Basel)
Weber A, Borghouts C, Brendel C, Moriggl R, Delis N, Brill B, Vafaizadeh V, Groner B.   +7 more
europepmc   +1 more source

BCR-ABL kinase domain mutations in CML patients, experience from a tertiary care center in North India. [PDF]

Leuk Res Rep
S A, Shah A, Ashish A, Kumar Singh N, Kaur M, Kumar Yadav A, Singh R.   +6 more
europepmc   +1 more source

FL118 Is a Potent Therapeutic Agent against Chronic Myeloid Leukemia Resistant to BCR-ABL Inhibitors through Targeting RNA Helicase DDX5. [PDF]

Int J Mol Sci
Takeda K, Ohta S, Nagao M, Kobayashi E, Tago K, Funakoshi-Tago M.   +5 more
europepmc   +1 more source

Chronic myeloid leukemia with two rare fusion gene transcripts of atypical BCR::ABL: A case report and literature review. [PDF]

Medicine (Baltimore)
Li Y, Zhang Y, Meng X, Chen S, Wang T, Zhang L, Ma X.   +6 more
europepmc   +1 more source

Editor's Note to: Targeting Primitive Chronic Myeloid Leukemia Cells by Effective Inhibition of a New AHI-1-BCR-ABL-JAK2 Complex. [PDF]

J Natl Cancer Inst
europepmc   +1 more source

Some of the next articles are maybe not open access.

Related searches:

CRKL Binding to BCR-ABL and BCR-ABL Transformation

Leukemia & Lymphoma, 1999
The SH2-SH3 domain-containing adaptor protein CRKL is the predominant tyrosine phosphorylated protein in chronic myelogenous leukemia (CML) neutrophils and BCR-ABL-expressing cell lines. The amino terminal CRKL SH3 domain binds directly to a proline-rich region in the C-terminus of BCR-ABL. BCR-ABL mutants with deletions of this region were constructed
K S, Kolibaba, A, Bhat, C, Heaney, T, Oda, B J, Druker   +4 more
openaire   +2 more sources

Biology of BCR-ABL

Blood Reviews, 1999
gene on chromosome 9 is the humanhomologue of a gene originally identified in a murineoncogenic virus, the Abelson murine leukaemia virus(A-MuLV). This retrovirus was isolated from a pred-nisolone treated mouse which developed lymphomafollowing innoculation with Moloney murineleukaemia virus (M-MuLV).
R, Chopra, Q Q, Pu, A G, Elefanty
openaire   +2 more sources

BCR/ABL and leukemia

Leukemia Research, 1996
This review focuses on the role of the chimeric BCR/ABL gene in leukemia development. First, we discuss and update knowledge regarding the molecular biology of BCR/ABL. We then review data regarding transforming activity of BCR/ABL. Third, we discuss the complex interactions between BCR/ABL and leukemia phenotype. We conclude with a brief discussion of
A, Butturini, R B, Arlinghaus, R P, Gale
openaire   +2 more sources

The BCR/ABL hybrid gene

Baillière's Clinical Haematology, 1987
A DNA region on chromosome 22, designated M-BCR, contains the chromosomal breakpoint of the Philadelphia (Ph) translocation in all Ph positive CML patients studied to date. M-BCR is part of a gene, BCR, oriented with its 5' end towards the centromere of chromosome 22. All of the CML DNAs analysed have a breakpoint within introns of the BCR gene.
J, Groffen, N, Heisterkamp
openaire   +2 more sources