The Grb2 binding site is required for the induction of chronic myeloid leukemia-like disease in mice by the Bcr/Abl tyrosine kinase [PDF]
Ryan P. Million, Richard A. Van Etten
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PBA2, a novel inhibitor of the β-catenin/CBP pathway, eradicates chronic myeloid leukemia including BCR-ABL T315I mutation. [PDF]
Yang K+8 more
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Differentiation of imatinib -resistant chronic myeloid leukemia cells with BCR-ABL-T315I mutation induced by Jiyuan Oridonin A. [PDF]
Xu Y+10 more
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Ontogeny of natural killer cells and T cells by analysis of BCR–ABL rearrangement from patients with chronic myelogenous leukaemia [PDF]
Eun Kyung Cho+8 more
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In Silico Molecular Modeling of Four New Afatinib Derived Molecules Targeting the Inhibition of the Mutated Form of BCR-ABL T315I. [PDF]
Rocha KML+3 more
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Are aberrant BCR–ABL transcripts more common than previously thought? [PDF]
G. A. Wilson+6 more
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Targeted degradation of oncogenic BCR-ABL by silencing the gene of NEDD8 E3 ligase RAPSYN. [PDF]
Sun Y+7 more
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