Results 201 to 210 of about 24,253,925 (274)

Liraglutide Ameliorates Injury‐Related Vascular Repair by Activating Autophagy Through Modulation of the PI3K–mTOR Signaling Pathway

open access: yesThe FASEB Journal, Volume 40, Issue 1, 15 January 2026.
Liraglutide inhibits chronic stress‐induced vascular injury in mice by inhibiting the activation of the PI3K‐mTOR signaling pathway and promoting autophagy of smooth muscle cells or macroautophagy. ABSTRACT Chronic stress is a significant risk factor for postoperative restenosis and other cardiovascular diseases, as it can promote the intimal ...
Zongyi Zhang   +14 more
wiley   +1 more source

LncRNA DANCR Activates p38/mTOR‐Mediated Autophagy via ANXA2 to Exacerbate Oxygen‐Induced Retinal Neovascularization in Mice

open access: yesThe FASEB Journal, Volume 40, Issue 1, 15 January 2026.
This study reveals that the long non‐coding RNA DANCR drives pathological retinal neovascularization in oxygen‐induced retinopathy via an ANXA2‐dependent p38/MAPK/mTOR autophagy pathway. In hypoxic endothelial cells and a murine OIR model, DANCR promotes angiogenesis.
Yini Wang, Jiale Bai, Shihong Zhao
wiley   +1 more source

Beclin 1 (BECN1) [PDF]

open access: yesScience-Business eXchange, 2009
openaire   +1 more source

Shenmai injection improves doxorubicin cardiotoxicity via miR-30a/Beclin 1.

open access: yesBiomedicine & pharmacotherapy = Biomedecine & pharmacotherapie, 2021
Xiaonan Zhang   +10 more
semanticscholar   +1 more source

Anti-Apoptotic and Anti-Oxidant Proteins in Glioblastomas: Immunohistochemical Expression of Beclin and DJ-1 and Its Correlation with Prognosis. [PDF]

open access: yes, 2019
Arcidiacono Biagio   +11 more
core   +1 more source

The Biological Role and Clinical Significance of BECLIN-1 in Cancer. [PDF]

open access: yesInt J Mol Sci
Maheshwari C   +6 more
europepmc   +1 more source

Epidermal growth factor receptor regulates Beclin-1 in hyperoxic acute lung injury. [PDF]

open access: yesBMJ Open Respir Res
Harris ZM   +23 more
europepmc   +1 more source

Home - About - Disclaimer - Privacy