Results 51 to 60 of about 1,382,457 (281)

A bio-inspired, motion-based analysis of crowd behavior attributes relevance to motion transparency, velocity gradients, and motion patterns. [PDF]

PLoS ONE, 2012
The analysis of motion crowds is concerned with the detection of potential hazards for individuals of the crowd. Existing methods analyze the statistics of pixel motion to classify non-dangerous or dangerous behavior, to detect outlier motions, or to ...
Florian Raudies, Heiko Neumann
doaj   +1 more source

Bio-Medical Library Bulletin, No. 19 (1971-03) [PDF]

, 1971
Bio-Medical Library. (1971). Bio-Medical Library Bulletin, No. 19 (1971-03).
Bio-Medical Library
core  

Longitudinal circulating tumor DNA profiling in patients with advanced endometrial cancer using an off‐the‐shelf targeted NGS panel

Molecular Oncology, EarlyView.
Intratumour heterogeneity complicates precision management of advanced endometrial cancer. Circulating tumor DNA (ctDNA) offers a minimally invasive strategy to capture tumor evolution and therapeutic resistance. Here, we compare tumor‐agnostic NGS with tumor‐informed ddPCR, outlining their relative sensitivity, concordance, and clinical implications ...
Carlos Casas‐Arozamena, Karin Teien Lande, Eva Diaz, Ana Vilar, Juan Cueva, Efigenia Arias, Victoria Sampayo, Alicia Abalo, Nerea González, Eva Colás, Antonio Gil‐Moreno, Miguel Abal, Gema Moreno‐Bueno, Therese Sørlie, Kristina Lindemann, Laura Muinelo‐Romay   +15 more
wiley   +1 more source

Bio-Medical Library Bulletin, No. 99 (1988-11) [PDF]

, 1988
Bio-Medical Library. (1988). Bio-Medical Library Bulletin, No. 99 (1988-11).
Bio-Medical Library
core  

Interaction of HS1BP3 with cortactin modulates TKS5 localisation, cell secretion and cancer malignancy

Molecular Oncology, EarlyView.
Here, we demonstrate that HS1BP3 interacts with Cortactin through a proline‐rich region (PRR3.1) and show that this interaction, and HS1BP3 itself, promote cancer cell proliferation and invasion. Inhibition of this interaction leads to build‐up of TKS5 in multivesicular endosomes and altered secretion of CD63 and CD9, providing an explanation for the ...
Arja Arnesen Løchen, Kristiane Søreng, Chiara Veroni, Laura Trachsel‐Moncho, Nagham Asp, Robin Gaupset, Lars Gustav Lyckander, Helene Knævelsrud, Lars Eftang, Anne Simonsen   +9 more
wiley   +1 more source

Investigation of nano-Bioconvective fluid motile microorganism and nanoparticle flow by considering MHD and thermal radiation

Informatics in Medicine Unlocked, 2020
In this study, the magnetohydrodynamic (MHD) flow in the presence of microorganisms and nanoparticles on a surface are investigated. The desired outcomes are motile microorganism number, Nusselt number, skin friction coefficient, and Sherwood number. The
Kh. Hosseinzadeh, Sajad Salehi, M.R. Mardani, F.Y. Mahmoudi, M. Waqas, D.D. Ganji   +5 more
doaj   +1 more source

Bio-Medical Library Bulletin, No. 91 (1986-10) [PDF]

, 1986
Bio-Medical Library. (1986). Bio-Medical Library Bulletin, No. 91 (1986-10).
Bio-Medical Library
core  

Developmental programmes drive cellular plasticity, disease progression and therapy resistance in lung adenocarcinoma

Molecular Oncology, EarlyView.
This study shows that lung adenocarcinomas exploit developmental branching morphogenesis to acquire a therapy resistant basal‐like tumour cell state. This process was found to be regulated by combined TP53 loss‐of‐function and type‐I interferon signalling, identifying a novel axis for biomarker and therapeutic target discovery.
Kamila J Bienkowska, Stephany Gallardo, Nur S. Zainal, Leena Arora, Matthew Ellis, Maria‐Antoinette Lopez, Judith Austine, Sai Pittla, Serena J Chee, Aiman Alzetani, Emily C Shaw, Christian H Ottensmeier, Gareth J Thomas, Christopher J Hanley   +13 more
wiley   +1 more source

Bio-Medical Library Bulletin, No. 112 (1993-10) [PDF]

, 1993
Bio-Medical Library. (1993). Bio-Medical Library Bulletin, No. 112 (1993-10).
Bio-Medical Library
core  

MITF maintains genome stability in nonmelanocyte lineages

Molecular Oncology, EarlyView.
MITF is essential for melanocyte survival and acts as an oncogene in 10%–20% of melanomas. We show that MITF depletion causes genome instability in nonmelanocytic cells, leading to LATS2‐mediated P53 activation, cell cycle arrest, and apoptosis. This study highlights the role of MITF as a genome maintenance factor beyond the melanocyte lineage. Created
Drifa H. Gudmundsdottir, Adrián López García de Lomana, Thejus B. Venkatesh, Kritika Kirty, Snaevar Sigurdsson, Linda Vidarsdottir, Ramile Dilshat, Erla Sveinbjornsdottir, Snaedis Ragnarsdottir, Daniel H Magnusson, Maria R. Bustos, Eirikur Steingrimsson, Thorkell Gudjonsson, Stefan Sigurdsson   +13 more
wiley   +1 more source